kuliah hipertensi 2013

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    HYPERTENSION

    Alfrina Hany

    Do You Know?

    The most common primary diagnosis in the United States, 50 million Americanaffected.

    Only 70% are aware they have HTN, only 50% are being treated.

    Only 25% of all hypertensive patients have their BP under control

    Cardiovascular risk increases two-fold for each 20mm/Hg rise in systolic pressure oreach 10mm/Hg rise in diastolic pressure

    Indonesiabest 3, 7% mortality

    Topics

    Definition

    Etiology

    Classification

    Clinical Manifestation

    Diagnostic Tests

    Complication

    What is Blood Pressure?

    The force of blood against the wall of the

    arteries.

    Systolic- as the heart beats

    Diastolic - as the heart relaxes

    Written as systolic over diastolic in mm Hg

    Williams & Hoppers, 2007

    Hypertension

    Hyperexcessive / highTensionPressure

    HypertensionHigh blood pressure

    Williams & Hopper, 2007

    Why is High Blood Pressure Important?

    Makes the Heart work too hard.Makes the walls of arteries hard.

    Increases risk for heart disease and

    stroke.

    Can cause heart failure, kidney

    disease, and blindness. Heart

    disease and stroke are the 1stand 3rd

    leading causes of death in the U.S.

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    Hypertension (HTN)

    is defined assustained abnormal

    elevation of the

    arterial blood

    pressure (Brashers,

    2006, p.1).

    Hypertension Defined Hipertensi

    keadaan bertahan dari kenaikan TD arterial

    sistemik, ditandai dengan kenaikan curah

    jantung /c.o dan tahanan periferal

    vaskular/svrmelibatkan multifaktorial

    Brunner & Suddarth, 2009

    70 bpm 70 mls

    4.9 L/min

    Cardiac outputFactors Controlling BP

    Etiology

    tidak dapat dihindari (heriditer, etnik, jenis kelamin, usia)

    dapat dihindari (gaya hidup, diet, alkohol, obesitas)

    penyakit penyebab (DM; CKD)

    Klasifikasi JNC VII (2003)

    (Brashers, 2006, p.1)

    Category Systol ic Diastol ic

    Normal 100

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    Klasifikasi berdasarkan tahapan

    High Blood Pressure

    Stage 1 140159 /9099

    Stage 2 160179 /100109

    Stage 3 180 /110

    Klasifikasi berdasarkan penyebab (1)

    Primary HypertensionIs usually of gradual onset

    Usually develops between the ages of30 and 50

    Tends to remain asymptomatic for 10to 20 years

    Essential or Primary-Underlyingpathophysiologic alteration of unknowncause

    95% of cases of HTN

    Contributing factors for Primary HTN:

    Psychological stress, high-sodium intake,and alcohol intake over 1 ounce per day

    Increased activity of: sympathetic nervous system (SNS) Renin-angiotensin-aldosterone system (RAA)

    Defects in natriuretic hormone function

    Inflammation

    Obesity

    Endothelial dysfunction

    Insulin resistance

    Klasifikasi berdasarkan penyebab (2)

    Secondary Hypertension

    Secondary-Resulting from a

    specific cause such as renal

    or endocrine disorders;

    Medications

    5% of cases

    Klasifikasi berdasarkan kegawatan

    Hipertensi emergensi

    TD diastolik>120 mmHg, kerusakan organ

    target, keterlambatankematian

    Hipertensi urgensi

    TD Diastolik > 120 mmHg, tanpa kerusakan

    organ, harus diturunkan 24 jam

    Manifestasi Klinis

    Silent killersymptoms freemild hypertension

    Kelelahan, konfusi, nausea, vomit, ansietas, keringat

    berlebihan, tremor, nyeri dada, epiktaksis,

    pandangan kabur, nokturia, azotemiasevere

    hypertension

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    Diagnostic Tests

    Laboratorium:

    HBnilai viskositas dan indikator faktor risiko (anemia, hiperkoagulability)Leukosit infeksi/kegagalan sumsum tulang

    BUN/Kreatininperfusi/faal ginjal

    Glukosahiperglikemi

    Kolesterolpredisposisi terbentuknya plak

    Serum aldosteronaldosteronisme primer

    Asam uratfaktor risiko

    Elektrolitkalium, natrium, kalsium

    Urineanalisa darah, protein, glukosa, identifikasi fungsi renal

    Diagnostic Tests

    Penunjang Lain:

    CT Scanemboli pada otakRontgen dadaedema paru, kardiomegali

    EKGkardiomegali, gangguan konduksi jantung

    Phaechromecytoma kolesterol dalam darah dan urin

    Echocardiogram: shows a graphic outline of the hearts movement

    Web of CausationGenetik,

    gerontologi,

    obesitas

    Perubahan

    struktur & Fgs PD

    Elastisitas PD

    Pompa jantung

    Aliran darah perifer

    Nikotin

    Merokok

    Asetil kolin ke PD

    Stress, cemas, takut

    Norepineprin

    Vasokonstriksi PD

    Tekanan sistemik

    epineprin

    Katekolamin

    DM

    Pelepasan renin

    Angiotensin I

    Angiotensin II

    Aldosteron

    Retensi Na & K

    Mediator nyeri

    PD Otak

    Hipertropi ventrikel

    Tekanan darah sistemik

    CO

    Beban jtg

    BUN & Cr

    Iskemi

    Nyeri kepala

    Vol.intravaskuler

    Ggn Perfusi Jar

    Filtrasi ginjal Kelebihan Vol.Cairan

    Penurunan CO

    Kelelahan

    Intoleransi

    aktivitas

    Nyeri akut/kronis

    HypertensionComplication

    Over long time, high bp damages arterial walls Sclerosis, decreased lumen

    Wall may dilate, tear Aneurysm

    Areas most frequently damaged: Kidneys, brain, retina

    End result of poorly controlled hypertension: Chronic kidney disease

    Stroke

    Loss of vision

    CHF

    How Does It Effect the Body?

    The Brain

    High blood pressure is the most important riskfactor for stroke.

    Can cause a break in a weakened blood vessel

    which then bleeds in the brain.

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    The Heart

    High Blood Pressure is a major risk factor for

    heart attack.Is the number one risk factor for Congestive

    Heart Failure.

    The Kidneys

    Kidneys act as filters to rid the body of wastes.

    High blood pressure can narrow and thicken theblood vessels.

    Waste builds up in the blood, can result in kidney

    damage.

    The Role of the Kidney

    Renin is made by the juxtaglomerular cells of the kidney

    Renin transforms plasma angiotensinogen toangiotensin I which is converted to angiotensin II by

    ACE

    Angiotensin II alters BP by increasing both PVR andblood volume by causing vasoconstriction and bystimulating the secretion of aldosterone which increasesthe reabsorption of sodium

    The Kidney cont.

    Angiotensinogen

    Renin

    Angiotensin I

    ACE vasoconstriction

    Angiotensin II Inc. PVR

    aldosterone (inc. reabsorp of Na)

    Inc. blood volume

    The Eyes

    Can eventually cause blood vessels to breakand bleed in the eye.

    Can result in blurred vision or even blindness.

    The Arteries

    Causes arteries to harden.

    This in turn causes the kidneys and heart to work

    harder.

    Contributes to a number of problems.

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    Life Style Changes

    Following a healthy eating pattern.

    Maintaining a healthy weight.

    Being Physically Active.

    Limiting Alcohol.

    Quitting Smoking.

    Penatalaksanaan non Farmakologi

    Tips for Reducing Sodium

    Buy fresh, plain frozen or canned no added salt

    veggies.

    Use fresh poultry, lean meat, and fish.

    Use herbs, spices, and salt-free seasonings at

    the table and while cooking.

    Choose convenience foods low in salt.

    Rinse canned foods to reduce sodium.

    Maintain Healthy Weight

    Blood pressure rises as weight rises.

    Obesity is also a risk factor for heartdisease.

    Even a 10# weight loss can reduceblood pressure.

    35-55% higher risk compared tothose maintaining ideal weight

    BMI > 30 kg / m2 or waist girth > 100cm (39.4 inches)

    Be Physically Active

    Helps lower blood pressure and lose/maintain weight.

    30 minutes of moderate level activity on

    most days of week. Can even break it

    up into 10 minute sessions.

    Use stairs instead of elevator, get off

    bus 2 stops early, Park your car at the

    far end of the lot and walk!

    Limit Alcohol Intake

    Alcohol raises blood pressure and canharm liver, brain, and heart

    What counts as a drink?

    12 oz beer

    5 oz of wine

    1.5 oz of 80 proof whiskey

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    Quit Smoking

    Injures blood vessel walls

    Speeds up process of hardening of thearteries.

    Current or quit within past 6 months

    Stimulate E & NE => raise HR +

    pheriperal vasoconstriction

    Farmakologi

    Vasodilator

    Mengembangkan PD

    arteri, kurangi

    tahanan perifer

    Diazoxide, Minoxidil,

    Prozasin

    Kalsium Antagonis

    Hambat masukan ion

    kalsium ke dalam sel

    dan turunkan

    afterload

    Diltiazem, Nifedipin

    Farmakologi

    ACE Inhibitor

    Hambat renin,

    angiotensin,

    vasodilatasi, turunkan

    vol.darah

    Captopril, Elanapril,

    Ramipril

    Diuretik

    Turunkan volume

    plasma, retensi

    perifer

    Furosemid, Thiazide

    Farmakologi

    Beta Bloker

    Kurangi impuls ke

    jantung dan PD

    Nervous system

    inhibitors

    slow nerve impulses

    to the heart.

    =ASKEP=Pengkajian

    Tn.K, Usia 55 thn

    Sangat pusing sekali

    Usaha harus tutup

    3 bulan ini pusing,

    jarang OR, sering

    makan malam hari

    TB 170cm, BB 93kg

    (BBI 63 kg, BMI 39,8)

    Pengusaha rumahmakan

    Nadi 60x/mnt regular

    TD190/110mmHg,T36,7

    Tangan dan kaki pucat

    terut.saat elevasi, kulit

    dingin dan lembab

    CRT ekstremitas 3 dtk

    Kolesterol total 245

    Askep=Analisa Data

    Usia 55 tahun

    Pengusaha RM

    Sering makan di

    malam hari

    TB 170cm, BB 93 kg

    BBI 63 kg, BMI

    Total serum kolesterol

    245 mg/dL.

    KetidakseimbanganNutrisi lebih dari

    kebutuhan tubuh

    intake makanan

    berlebihan akibat

    kebutuhan metabolik

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    Askep=Analisa Data

    Sering makan di

    malam hari

    Jarang berolahraga

    Sebelumnya merasa

    penyakit belum parah

    sehingga tidak perlu

    periksa

    Inefective health

    maintenancekopingindividu yang tidak

    efektif dengan gaya

    hidup yang tidak sehat

    Askep=Analisa Data

    Mengatakan sakit kepala

    Nadi 60x/mnt regular, TD(duduk): 190/110 mmHg

    Tangan dan kaki pucat

    terutama saat elevasi,

    CRT ekstremitas 3 detik

    Kulit teraba dingin dan

    lembab

    Ketidakefektifan perfusi

    jaringan periferproses penyakit

    (hipertensi)

    Askep=Analisa Data

    Sangat sibuk dengan

    usahanya

    Kadang pusing 3

    bulan ini tapi tidak

    pernah periksa

    Klien suka makanan

    berlemak dan merasahal tersebut biasa

    dalam keluarganya

    Kurang Pengetahuan

    kurangnya paparan

    informasi

    Askep=Analisa Data

    Kepala sangat pusing

    sekali

    Ketidaknyamanan

    proses penyakit

    DAFTAR DIAGNOSA KEP

    KetidakseimbanganNutrisi lebih dari

    kebutuhan tubuh

    intake makanan

    berlebihan

    Inefective health

    maintenance

    (ketidakefektifan

    pemeliharaan

    kesehatan)gaya

    hidup tidak sehat

    Ketidakefektifan perfusijaringan perifer

    hipertensi

    Kurang pengetahuan

    kurang informasi tentang

    pengobatan

    Risiko penurunan perfusi

    jaringan jantung

    Ketidaknyamanan

    proses penyakit (HT)

    Diagnosa 1 Nutrisi

    Dalam 4 minggu,kelebihan nutrisi klien

    dapat mencapai

    indikator 3 pada

    NOC : nutritional

    status

    Intake nutrient : diet seimbang : KH, Prot,Lemak, Mineralindikator 3

    Food intake : DASH bertahap mulai diet

    HT 1indikator 3

    Energy : olahraga 30 menit sehari

    indikator 3

    Weight/height ratio : BBI dan BMI

    menurun bertahapindikator 3

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    Diagnosa 1 intervensi

    Domain: Nutrition

    support

    Nutrition management

    1.1 Identifikasi pilihan

    makan klien

    1.2 anjurkan intake

    kalori yang sesuai

    2.1 yakinkan perlunya

    diet DASH

    2.2 kolaborasi diet

    DASHdiet HT

    sesuai klien

    Conclusion

    Cardiovascular disease is the number one killer.Hypertension is a very controllable disease, withdrastic consequences if left uncontrolled.

    It is highly preventable and controllable with dietand exercise.

    Good resource: www.americanheart.org;www.nhlbi.nih.gov

    Thanks

    Good Luck and see you around

    http://www.americanheart.org/http://www.nhlbi.nih.gov/http://www.nhlbi.nih.gov/http://www.americanheart.org/