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1. Pendidikan Kesehatan

SATUAN ACARA PENYULUHAN

(SAP)

Tema

: Penyakit Cor Pulmonal Sub Tema

: Perawatan Penyakit Cor PulmonalSasaran

: Bp. XTempat

: Rumah Sakit HHari/Tanggal

: Jumat, 5 Desember 2012Waktu

: 30 Menit

A. Tujuan Instruksional Umum

Setelah mengikuti penyuluhan selama 30 menit, diharapkan Bp. H dapat menjelaskan penyakit Cor Pulmonal.

B. Tujuan Instruksional Khusus

Setelah mengikuti penyuluhan selama 30 menit, diharapkan Klien dapat:

1. Menjelaskan pengertian penyakit Cor Pulmonal dengan benar

2. Menyebutkan faktor penyebab yang dapat menimbulkan penyakit Cor Pulmonal3. Menyebutkan tanda/gejala dari penyakit Cor Pulmonal4. Menjelaskan penatalaksanaan penyakit Cor Pulmonal5. Menjelaskan patofisiologi penyakit Cor PulmonalC. Materi

1. Pengertian penyakit Cor Pulmonal2. Faktor penyebab dari penyakit Cor Pulmonal3. Tanda/gejala penyakit Cor Pulmonal4. Penatalaksanaan penyakit Cor Pulmonal5. Patofisiologi penyakit Cor PulmonalD. Metode

1. Ceramah

2. Tanya jawab

E. Kegiatan Penyuluhan

NoKegiatanPenyuluhPesertaWaktu

1.Pembukaan Salam pembuka

Menyampaikan tujuan penyuluhan

Menjawab salam

Menyimak,

Mendengarkan, menjawab pertanyaan5 Menit

2.Kerja/ isi Penjelasan pengertian, penyebab, gejala, penatalaksanaan dan patofisiologi penyakit Cor Pulmonal Memberi kesempatan peserta untuk bertanya

Menjawab pertanyaan

Evaluasi Mendengarkan dengan penuh perhatian Menanyakan hal-hal yang belum jelas

Memperhatikan jawaban dari penceramah Menjawab pertanyaan 15 menit

3.Penutup Menyimpulkan

Salam penutup Mendengarkan

Menjawab salam10 Menit

F. Media

Leaflet: Tentang penyakit Cor Pulmonal

G. Sumber/Referensi

a. Doenges, E. Marilynn. 2002. Rencana Asuhan Keperawatan Ed. 3. EGC : Jakarta.

b. FKUI. 1999. Buku Ajar Ilmu Penyakit Dalam jilid 1. FKUI : Jakarta.H. Evaluasi

Formatif:

1. Klien dapat menjelaskan pengertian penyakit Cor Pulmonal2. Klien mampu menjelaskan faktor penyebab dari penyakit Cor Pulmonal3. Klien dapat menjelaskan tanda/gejala penyakit Cor Pulmonal4. Klien mampu menjelaskan penatalaksanaan penyakit Cor Pulmonal

5. Klien mampu menjelaskan Patofisiologi penyakit Cor PulmonalSumatif:

Klien dapat memahami penyakit Cor PulmonalYogyakarta, 5 Desember 2012Pembimbing

Penyuluh

(Diah Pujiastuti, S. Kep., Ns)

(Ni Gusti Ayu Kadek D.)2. Jurnal

The association between obesity, mortality and filling pressures in pulmonary hypertension patients; the "obesity paradox"

Zafrir B, Adir Y, Shehadeh W, Shteinberg M, Salman N, Amir O.

A. Source

Department of Cardiovascular Medicine, Lady Davis Carmel Medical Center, Haifa, Israel; The Heart Failure Center, Lin medical Center, Haifa, Israel. Electronic address: barakzmd @ gmail. com.

B. Abstract

BACKGROUND:

The term "obesity paradox", refers to lower mortality rates in obese patients, and is evident in various chronic cardiovascular disorders. There is however, only scarce data regarding the clinical implication of obesity and pulmonary hypertension (PH). Therefore, in the current study, we evaluated the possible prognostic implications of obesity in PH patients.

METHODS:

We assessed 105 consecutive PH patients for clinical and hemodynamic parameters, focusing on the possible association between Body Mass Index (BMI) and mortality. Follow-up period was 1913 months.

RESULTS:

Sixty-one patients (58%) had pre-capillary PH and 39 patients (37%) out-of-proportion post-capillary PH. During follow-up period, 30 patients (29%) died. Death was associated with reduced functional-class, inverse-relation with BMI, higher pulmonary artery and right atrial pressures, pulmonary vascular resistance and signs of right ventricular failure. In multivariate analysis, obesity (BMI30kg/m), was the variable most significantly correlated with improved survival [H.R 0.2, 95% C.I 0.1-0.6; p=0.004], even after adjustment for baseline characteristics. Obese and very-obese (BMI35kg/m) patients had significantly less mortality rates during follow-up (12% and 8%, respectively) than non-obese patients (41%), p=0.01. The tendency of survival benefit for the obese vs. non-obese patients was maintained both in the pre-capillary (10% vs. 46% mortality, p=0.008) and disproportional post-capillary PH patients (11% vs. 40% mortality, p=0.04).

CONCLUSIONS:

Obesity was significantly associated with lower mortality in both pre-capillary and disproportional post-capillary PH patients. It seems that in PH, similarly to other chronic clinical cardiovascular disease states, there may be a protective effect of obesity, compatible with the "obesity paradox".

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