trigger number five
TRANSCRIPT
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Trigger number five
Ayu Kartika Sari
405080161
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Rhinitis Alergi
Penyakit inflamasi yg disebabkan oleh
reaksi alergi pd pasien atopi yg
sebelumnya sudah tersensitisasi dgn
alergen yg sama serta dilepaskannya
suatu mediator kimia ketika terjadi
paparan ulangan dgn alergen spesifik tsb.
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Klasifikasi
Saat ini digunakan ygberdasarkan klasifikasi WHO,
ada 2 yaitu:a. Intermitten(kadang): gejala
4hari/mgg ATAU > 4mgg
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Manifestasi klinis
Gejala khasnya adalah serangan bersin
berulang
Keluar ingus/rinore yg encer&banyak
Hidung tersumbat
Hidung dan mata gatal, kadang disertai
banyak air mata
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Komplikasi
Polip hidung
Otitis media efusi yg sedang residif,
terutama pada anak2
Sinusitis paranasal
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Sinusitis
Inflamasi mukosa sinus paranasal (sinus
maksilaris, ethmoidalis, sphenoidalis,
frontalis)
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Etiologi
Virus
Rhinitis terutama Rhinitis alergi
Rhinitis hormonal pd wanita hamil
Polip hidung Kelainan anatomi seperti deviasi septum/ hipertrofikonka
Infeksi tonsil
Infeksi gigi Sumbatan KOM
Kelainan imunologik
Diskinesia silia
Penyakit fibrosis kistik
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Faktor predisposisi
Lingkungan berpolusi
Udara dingin dan kering
Kebiasaan merokok
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Manifestasi klinis
Keluhan utama(akut)= HIDUNG TERSUMBAT
Nyeri di daerah sinus
Sakit kepala
Hiposmia/anosmia
Halitosis
PND yg menyebabkan batuk dan sesak pdanak
Sinusitis kronik tidak khas gejalanya.
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Pemeriksaan
Yg terpenting adalah menggunakan CT
Scan
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Penatalaksanaan
Antibiotikuntuk sinusitis kronik
Dekongestan oral dan topikal
Antihistamin tidak rutin diberikan
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Komplikasi
Kelainan orbita
Kelainan intrakranial
Osteomilitis dan abses subperiostal Kelainan paru
P k Eti l i G j l Kh P ik T t L k
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Penyak
it
Etiologi Gejala Khas Pemeriksaan
Penunjang
Tata Laksana
Deviasi
Septum
Trauma/
ketidakseimb
angan
pertumbuhan.
Sumbatan hidung yang
menetap, Pada sisi deviasi
terdapat konka hipotrofi,
sedangkan sebelahnya terjadi
konka hipertrofi akibat
kompensasi.
Pada pemeriksaan terlihat
rongga hidung tidak sama
besarnya antara kanan dan kiri.
Penonjolan tulang/ rawan
septum berbentuk krista/ spina/perlekatan
- Tanpa gejala/
keluhan sangat
ringan tidak perlu
koreksi.
Keluhan nyata
dirujuk untuk
dilakukan reseksi
submukosa atau
septoplasti (reposisi
septum).
Polip
Nasi
Akibat reaksi
hipersensitif
atau reaksi
atopik di
dalam selaputmukosa
hidung
Sumbatan hidung yang
menetap, makin lama makin
berat.
Hiposmia/ anosmia.
Tampak massa putih keabu-abuan/ kuning kemarahan dalam
kavum nasi.
Polip bertangkai sehingga
mudah digerakkan,
konsistensinya lunak, tidak nyeri
tekan, tidak mudah berdarah,
dan tidak mengecil padapemakaian vasokonstriktor.
Rontgen
sinus.
Pembuatan
biakan hapus
dari hidung(sebelum
melakukan
pengeluaran
polip).
Bila polip kecil,
dapat diobati secara
konservatif dengan
kortikosteroid
sistemik/ oral/ topikal.Bila polip besar,
dilakukan ekstrasi
polip dengan senar.
Bila berulang dapat
dirujuk untuk operasi
etmoidektomi
intranasal/ekstranasal.
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Faringitis
Peradangan akut membran mukosa faring
dan struktur lain di sekitarnya
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Gejala
Nyeri tenggorokan
Mual muntah
Faring hiperemis
Demam
Nyeri tenggorokan
Tonsil bengkak
KGB leher anterior bengkak dan nyeri
Uvula bengkak dan merah
Petekie palatum mole
Ruam skarlatina
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Klasifikasi
Saya hanya membahas faringitis akut
karena kasus kita juga ttg faringitis
akut(karena belum lebih dari 14 hari):
>>Faringitis viral
>>Faringitis bakterial
>>Faringitis fungal
J i P b b G j l t d T i
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Jenis
Faringitis
Penyebab
Tbanyak
Gejala tanda Terapi
F.VIRAL Rhinovirus -Demam,
-rinorea,
-mual,-nyeri tenggorok
-susah mnelan
Istirahat,
minum yg cukup,
kumur air hangat,analgetika&tablet hisap,
AV metisoprinol pd infeksi
herpes simpleks
F.BAKTERIAL Strepto
kokus beta
hemolitikus
group A
-Nyeri kepala hebat,
-muntah,
-deman dgn suhu
tinggi,
-JARANG dgn BATUK
antibiotik(penicilin),
kostikosteroid,
analgetika,
kumur dgn air
hangat/antiseptik
F.FUNGAL Candida -nyeri tenggorok dannyeri mnelan
-Mukosa faring
hiperemis
Nystatin,
analgetika
F.GONOREA Kontak
orogenital -Sefalosporin generasi ke-3,
Ceftriakson IM
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Pneumonia
Peradangan parenkim paru, distal dari
bronkiolus terminalis yg mencakup
bronkiolus respiratorius, dan alveoli serta
menimbulkan konsolidasi jaringan parudan gangguan pertukaran gas setempat
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Etiologi
Berdasarkan kuman/ virus
Berdasarkan lokalisasi
Berdasarkan keadaan penderita
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Klasifikasi
Pneumonia di rumah perawatan(PN)
Pneumonia yg terjadi >48jam setelah
dirawat di RS tapi tidak sedang memakai
ventilator
Pneumonia Komunitas(PK)
Pneumonia akibat infeksi di luar RS
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Manifestasi Klinis
Batuk dgn napas cepat
Hidung melebar
grunting Demam
Ada crackles(ronki basah)
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Faktor resiko PK
Usia >65thn
Adanya infeksi paru yg
multilober/nekrotikans, pasca obstruksi
atau aspirasi
Penyakit penyerta(PPOK, DM,
bronkiektasis, gagal ginjal, malnutrisi, dll)
Tanda fisik yg memprediksi mortalitas,
morbiditas dan komplikasi
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Pemeriksaan
Gambaran radiologis berupa pneumonia
alveolar dgn gambaran air bronkogram
Distribusi infiltrat pd segmen apikal lobus
bawah atau inferior lobus
Bentuk lesi berupa kavitasi dgn air fluid
level sugestif
Pembentukan kista
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Leukositosis
Titer antibodi terhadap virus, legionella,
dan mikoplasmatiter tinggi/ kenaikan
titer 4x
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Penatalaksanaan
Bacterial. Doctors usually treat bacterial pneumonia withantibiotics. Stopping medication too soon may cause yourpneumonia to return. It also helps create strains of bacteriathat are resistant to antibiotics.
Viral. Antibiotics aren't effective against most viral forms of
pneumonia. And although a few viral pneumonias may betreated with antiviral medications, the recommendedtreatment generally is rest and plenty of fluids.
Mycoplasma. Mycoplasma pneumonias are treated withantibiotics. Many cases of mycoplasma pneumonia goundiagnosed and untreated. The signs and symptoms mimic
those of a bad chest cold, so some people never seekmedical attention. The symptoms generally go away on theirown.
Fungal. If your pneumonia is caused by a fungus, you'll likelybe treated with antifungal medication.
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Komplikasi
Bacteria in your bloodstream. Pneumonia can be life-threatening wheninflammation from the disease fills the air sacs in your lungs and interfereswith your ability to breathe. In some cases the infection may invade yourbloodstream (bacteremia). It can then spread quickly to other organs.
Fluid accumulation and infection around your lungs. Sometimes fluidaccumulates between the thin, transparent membrane (pleura) coveringyour lungs and the membrane that lines the inner surface of your chest wall a condition known as pleural effusion. Normally, the pleurae are smooth,allowing your lungs to slide easily along your chest wall when you breathe inand out. But when the pleurae around your lungs become inflamed(pleurisy) often as a result of pneumonia fluid can accumulate andmay become infected (empyema).
Lung abscess. A cavity containing pus (abscess) that forms within the areaaffected by pneumonia is another potential complication.
Acute respiratory distress syndrome (ARDS). The pneumonia involvesmost areas of both lungs, making breathing difficult and depriving your bodyof oxygen. Underlying lung disease of any kind, but especially COPD,makes you more susceptible to ARDS.
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TBC
Tuberkulosis (TBC atau TB) adalah suatu
penyakit infeksi yang disebabkan oleh
bakteri Mikobakterium tuberkulosa. Bakteri
ini merupakan bakteri basil yang sangatkuat sehingga memerlukan waktu lama
untuk mengobatinya. Bakteri ini lebih
sering menginfeksi organ paru-parudibandingkan bagian lain tubuh manusia.
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Epidemiologi
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Etiologi
Mycobacterium tuberculosis, sejenis
kuman berbentuk batang dgn panjang 1-
4/um dan tebal 0,3-0,6/um
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Manfes
Batuk terus-menerus selama 3minggu/ lebih Sakit di dada
Batuk berdarah atau berdahak (lendir daribagian dalam paru2)
Letih/ mudah capai
BB
Kehilangan nafsu makan
Demam Menggigil
Berkeringat di malam hari
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Pemeriksaan Penunjang
*A small reaction (5 mm of hard swelling at the site) is considered to be positive inpeople:-Who have HIV-Who are taking steroid therapy-Who have been in close contact with a person who has active tuberculosis*Larger reactions (greater than or equal to 10 mm) are considered positive in:-People with diabetes or kidney failure-Health care workers*In people with no known risks for tuberculosis, a positive reaction requires 15 mm
or more of hard swelling at the site
Purified ProteinDerivative(PPD) adalah
sebuah antigen yg
disuntikan secara
intradermal di bagian
volar lengan bawah.
Setelah 48-72 jam,bagian yg disuntik akan
dievaluasi oleh dokter.
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P t l k IMUNISASI KIE
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Penatalaksanaan IMUNISASI,KIE,
FARMAKO
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Tumor paru
Tipe histologi:
1. Small cell ---------------------20%
2. Squamus carcinoma ---------30%.3. Adenokarsinoma ------------40%
4. Large cell carcinoma --------10%.
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Small cell carcinoma :
- Bersifat agresif, mudah bermetastase.
- Respons terhadap kemoterapi cukup baik.
- Mikroskopik:* Kelompokan sel hiperkromatik dengan inti
moulding (secara sitologi).
* Sel spindle,inti gelap.
* Tersusun berkelompok (sheet, cord).
- Sel tumbuh dari sel neuroendokrine
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Adenokarsinoma. :
- Sering pada wanita.
- Non smokers.
- 2/3 dijumpai di daerah perifer paru.
- Ada 2 jenis:
1. Sel tumor tersusun bentuk acini
dengan jaringan ikat stroma.
2. Sel tumor tersusun sepanjangdinding alveolus ( bronchi alveolar
carcinoma).
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Squamus cell carcinoma :
Hubungan dekat dengan merokok
Tumbuh dari bronchi besar dengan epitel
yang mengalami metaplasi displasi
Sering ada perdarahan dan nekrosis
Berdiferensiasi baik jelek
Metastase ke kelenjar Hilus
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Large cell carcinoma :
- Tumbuh di sentral.
- Sel besar.- Etiologi :
* Rokok : - aktif
-pasifkarena adanya benzypyrene
dan nitrosamine.
* Polusi : industri, asbes, radiasi,
bahan metal.
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Bronchiechtasis
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SALURAN NAPAS BAWAH
BAGIAN
OBSTRUKSI
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Bronkiolitis
swelling and mucus buildup in the smallestair passages in the lungs (bronchioles),
usually due to a viral infection.
usually affects children under the age of 2,with a peak age of 3 - 6 months
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Etiologi
Respiratory syncytial virus (RSV) is the
most common cause.
Other viruses that can cause bronchiolitis
include:
Adenovirus
Influenza
Parainfluenza
http://www.nlm.nih.gov/medlineplus/ency/article/001564.htmhttp://www.nlm.nih.gov/medlineplus/ency/article/000080.htmhttp://www.nlm.nih.gov/medlineplus/ency/article/001370.htmhttp://www.nlm.nih.gov/medlineplus/ency/article/001370.htmhttp://www.nlm.nih.gov/medlineplus/ency/article/000080.htmhttp://www.nlm.nih.gov/medlineplus/ency/article/001564.htm -
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Manifestasi Klinis
Bluish skin due to lack of oxygen (cyanosis) Cough, wheezing, shortness of breath, or
difficulty breathing
Fever
Intercostal retractions
Nasal flaring in infants
Rapid breathing (tachypnea)
Decreased blood oxygen Wheezing and crackling sounds heard
through stethoscope exam of chest
http://www.nlm.nih.gov/medlineplus/ency/article/003215.htmhttp://www.nlm.nih.gov/medlineplus/ency/article/003070.htmhttp://www.nlm.nih.gov/medlineplus/ency/article/003070.htmhttp://www.nlm.nih.gov/medlineplus/ency/article/003215.htm -
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Penatalaksanaan
Sometimes, no treatment is necessary. Supportive therapy can include:
-Chest clapping
-Drinking enough fluids. Breast milk or formula are okay for childrenyounger than 12 months. Offer warm lemonade or apple juice if yourchild is over 4 months.
-Breathing moist (wet) air helps loosen the sticky mucus that may bechoking your child. You can use a humidifier to moisten the air yourchild is breathing. Follow the directions that come with thehumidifier.
-Getting plenty of rest
-Do not let anyone smoke in the house, car, or anywhere near your
child.*Antibiotics are not effective against viral infections. Mostmedications have little effect on bronchiolitis. Children in the hospitalmay need oxygen therapy and fluids given through a vein (IV) tostay hydrated.
-In extremely ill children, antiviral medications (such as ribavirin) are
used in rare cases.
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Komplikasi
Airway disease, including asthma, later in
life
Respiratory failure
Additional infection, such as pneumonia
http://www.nlm.nih.gov/medlineplus/ency/article/000145.htmhttp://www.nlm.nih.gov/medlineplus/ency/article/000145.htm -
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Faktor Resiko
Exposure to cigarette smoke
Age younger than 6 months old
Living in crowded conditions
Lack of breast-feeding
Prematurity (being born before 37 weeks
gestation)
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COPD
Chronic obstructive pulmonary disease (COPD)
refers to chronic lung disorders that result in
blocked air flow in the lungs. The two main
COPD disorders are emphysema and chronic
bronchitis, the most common causes of
respiratory failure.
Emphysema occurs when the walls between
the lung's air sacs become weakened and
collapse. Damage from COPD is usually
permanent and irreversible.
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Chronic bronchitis is most frequently causedby long term irritation of the bronchial tubes.
Bronchitis is considered "chronic" if symptoms
continue for three months or longer. Bronchitis
caused by allergies can also be classified as
chronic bronchitis. Chronic bronchitis is caused
most often by exposure to airborne pollutantssuch as cigarette smoke, excessive dust in the
air, or chemicals. The bronchial lining becomes
inflamed and the constant exposure to such
pollutants begins to cause damage in the
bronchioles (the smaller airways in the lungs).
Symptoms of chronic bronchitis includeshortness of breath or wheezing, chest pain,
and chronic productive cough.
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Asthma
Asthma is a lung disease that makes itharder to move air in and out of yourlungs. There are three things that you
should know about asthma: Asthma is chronic. In other words, you live
with it every day.
It can be serious even life threatening.
There is no cure for asthma, but it can bemanaged so you live a normal, healthy life.
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Etiology
Genetics. Asthma tends to runs in families. Genetics plays animportant role in causing asthma. If your mom or dad have asthma,then you are more likely to have asthma too.
Allergies. Some people are more likely to develop allergies thanothers, especially if your mom or dad had allergies. Certain allergiesare linked to people who get asthma.
Respiratory Infections. As the lungs develop in infancy and earlychildhood, certain respiratory infections have been shown to causeinflammation and damage the lung tissue. The damage that iscaused in infancy or early childhood can impact lung function long-term.
Environment. Contact with allergens, certain irritants, or exposure to
viral infections as an infant or in early childhood when the immunesystem in developing have been linked to developing asthma.
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Predisposition factors
Respiratory Infections. such as a cold, flu, or sinus infection, are the mostcommon cause of asthma symptoms leading to an asthma flare-up
Allergens. Allergens, such as pollen, mold, dust, dust mites, cockroaches,animal droppings, and dander from animals with fur or feathers
Irritants. include perfumes, cleaning fluids, cigarette smoke, air pollution,wood smoke and kerosene heaters.
Exercise. They may produce more mucus leading to asthma symptoms. Emotions. Crying, yelling or even laughing may cause asthmasymptoms. Stress, both personal and work-related, can be a major trigger.
Chemicals. Certain chemicals cause severe asthma symptoms in peoplewith asthma. Seasonal/Time Triggers. Some people with asthmaexperience more problems with symptoms at certain times of the day(during sleeping time when airways tend to close to close some), certain
times of the month (hormonal changes in women during pregnancy ormenstruation), or certain times of the year (when pollen levels are high suchas in the fall and the spring).
Smoking. Tobacco smoke irritates the airways.
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Manifestasi klinis
Wheezing. You may notice a wheezing soundwhen you breathe. Sometimes this happens onlywhen you exercise or have a cold.
Frequent Cough. This may be more common at
night. You may or may not cough up mucus. Shortness of Breath. This is the feeling you can't
get enough air into your lungs. It may occur onlyonce in a while, or often.
Chest Tightness. Your chest may feel tight,especially during cold weather or exercise. Thiscan also be the first sign of a flare-up.
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PATOFISIOLOGI
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Pencegahan
Get a flu shot. Protect your infants and toddlers
from getting the flu. Children 6 months of age
and older are one of the targeted group to
receive the influenza vaccine. If you care for achild less than 6 months of age, it important for
you to get a flu shot to help protect them from
getting sick.
Avoid exposure to tobacco smoke. If you or afamily member smokes, don't smoke around
your child. Make a plan to quit
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Farmako asthma
Beta adrenoceptor agonist
Theophyline
Corticosteroids
Chromolyn dan Nedocromil
Leukotrien pathway inhibitor
Anti Ig-E monoclonal antibody
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PERBEDAAN PPOK DAN ASMA
PPOK Asma
Umur mula Dekade ke 6 Variasipenyakit paling cepat awal 40
Peran Sangat berperan - kurang berperanmerokok - dpt memperberat
Reversibiliti obstruksi kronik - obs. episodikobstruksi dan menetap - VEP-1 normal
waktu remisiVEP-1 menetap - VEP-1 membaikdgn bronkodilator dgn bronkodilatorsedikit perbaikan
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PPOK Asma
Perjalanan progresif episodikpenyakit lambat
Riwayat jarang sering
alergi
Kapasiti menurun normaldifusi
Hipoksemia kronik jarang
Spirometri dapat membaik perbaikan nyatadgn bronkodilator dgn bronkodilator