syok fk umsu 2012

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    Diagnosis dan Manajemen

    Syok

    Diagnosis dan Manajemen

    Syok

    Irfan Hamdani

    Dept. Anestesiologi FK UMSU

    Irfan Hamdani

    Dept. Anestesiologi FK UMSU

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    SHK 2

    Shock

    Shock

    • Alwas a smptom of primar ca!se

    • Inade"!ate #lood flow to meet tiss!e

    o$gen demand• Ma #e associated with hpotension

    • Associated with signs of hpoperf!sion%

    mental stat!s change& olig!ria& acidosis

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    SHK '

     

    Kategori Syok 

    Kategori Syok

    • (ardiogenic

    • Hpo)olemic

    • Distri#!ti)e

    • *#str!cti)e

    • (ardiogenic

    • Hpo)olemic• Distri#!ti)e

    • *#str!cti)e

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    SHK +

    (ardiogenic Shock

    (ardiogenic Shock

    • kontraktilitas ,ant!ng

    men!r!n

    • -eningkatan tekanan

    pengisian ,ant!ng&

    pen!r!nan S&

    pen!r!nan (*&

    peningkatan S/

    • kontraktilitas ,ant!ng

    men!r!n

    • -eningkatan tekanan

    pengisian ,ant!ng&

    pen!r!nan S&

    pen!r!nan (*&

    peningkatan S/

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    SHK 0

    Hpo)olemic Shock

    • Decreased cardiac o!tp!t

    • Decreased filling press!res

    • (ompensator increase in

    sstemic )asc!lar resistance

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    SHK 1

    Distri#!ti)e ShockDistri#!ti)e Shock

    • ormal or increased cardiac o!tp!t

    • 3ow sstemic )asc!lar resistance

    • 3ow to normal filling press!res

    • Sepsis& anaphla$is& ne!rogenic&

    and ac!te adrenal ins!fficienc

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    SHK 4

    *#str!cti)e Shock*#str!cti)e Shock

    • Decreased cardiac o!tp!t

    • Increased sstemic )asc!lar

    resistance

    • aria#le filling press!resdependent on etiolog

    • (ardiac tamponade& tension

    pne!mothora$& massi)e

    p!lmonar em#ol!s

    • Decreased cardiac o!tp!t

    • Increased sstemic )asc!lar

    resistance

    • aria#le filling press!resdependent on etiolog

    • (ardiac tamponade& tension

    pne!mothora$& massi)e

    p!lmonar em#ol!s

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    SHK 5

    (ardiogenic Shock Management(ardiogenic Shock Management

    • 6reat arrhthmias

    • Diastolic dsf!nction ma

    re"!ire increased filling

    press!res

    • asodilators if not hpotensi)e

    • Inotrope administration

    • 6reat arrhthmias

    • Diastolic dsf!nction ma

    re"!ire increased filling

    press!res

    • asodilators if not hpotensi)e

    • Inotrope administration

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    SHK 7

    (ardiogenic Shock Management

    • asopressor agent needed if

    hpotension present to raise

    aortic diastolic press!re

    • (ons!ltation for mechanicalassist de)ice

    • -reload and afterload red!ction

    to impro)e hpo$emia if #lood

    press!re ade"!ate

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    SHK 89

    Hpo)olemic Shock

    Management

    Hpo)olemic Shock

    Management

    • ol!me res!scitation : crstalloid&

    colloid

    • Initial crstalloid choices

     – 3actated /inger;s sol!tion

     – ormal saline lood& crstalloid& colloid

    • ol!me res!scitation : crstalloid&

    colloid

    • Initial crstalloid choices

     – 3actated /inger;s sol!tion

     – ormal saline lood& crstalloid& colloid

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    SHK 88

    Distri#!ti)e Shock 6herap

    • /estore intra)asc!lar )ol!me

    • Hpotension despite )ol!me therap

     – Inotropes and?or )asopressors

    • asopressors for MA- @ 19 mm Hg

    • Ad,!ncti)e inter)entions dependent on

    etiolog

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    SHK 82

    *#str!cti)e Shock 6reatment*#str!cti)e Shock 6reatment

    • /elie)e o#str!ction

     – -ericardiocentesis

     –6!#e thoracostom

     – 6reat p!lmonar em#ol!s

    • 6emporar #enefit from fl!id

    or inotrope administration

    • /elie)e o#str!ction

     – -ericardiocentesis

     – 6!#e thoracostom

     – 6reat p!lmonar em#ol!s

    • 6emporar #enefit from fl!id

    or inotrope administration

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    SHK 8'

    Fl!id 6herapFl!id 6herap

    • (rstalloids – 3actated /inger;s sol!tion

     – ormal saline

    • (olloids

     – Hetastarch

     – Al#!min

     – elatins

    • -acked red #lood cells• Inf!se to phsiologic endpoints

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    SHK 8+

    Fl!id 6herap

    • (orrect hpotension first

    • Decrease heart rate

    • (orrect hpoperf!sion a#normalities• Monitor for deterioration of

    o$genation

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    SHK 80

    Inotropic ? asopressor Agents

    • Dopamine

     – 3ow dose

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    SHK 81

    Inotropic Agents

    • Do#!tamine

     – 0B29µ

    g?kg?min

     – Inotropic and )aria#le chronotropic effects

     – Decrease in sstemic )asc!lar resistance

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    SHK 84

    Inotropic ? asopressor

    Agents• orepinephrine

     – 9.90 µg?kg?min and titrate to effect

     – Inotropic and )asopressor effects

     – -otent )asopressor at high doses

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    SHK 85

    Inotropic ? asopressor Agents

    • pinephrine

     – >oth and β actions for inotropic

    and )asopressor effects

     – 9.8µ

    g?kg?min and titrate

     – Increases mocardial *2 cons!mption

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    SHK 87

      6herape!tic oals in Shock 6herape!tic oals in Shock

    • Meningkatkan deli)eri *2

    • Mengoptimalkan *2 di darah

    •  meningkatkan cardiac o!tp!t and tekanan

    darah• Menes!aikan ke#!t!han *2 sistemik dan

    hantaran *2

    •  Mencegah organ hpoperf!sion

    • Meningkatkan deli)eri *2

    • Mengoptimalkan *2 di darah

    •  meningkatkan cardiac o!tp!t and tekanan

    darah• Menes!aikan ke#!t!han *2 sistemik dan

    hantaran *2

    •  Mencegah organ hpoperf!sion

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    SHK 29

    *lig!ria

    • Marker of hpoperf!sion• Urine o!tp!t in ad!lts

    @9.0 m3?kg?hr for C2 hrs

    • tiologies – -rerenal

     – /enal

     – -ostrenal

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    SHK 28

    )al!ation of *lig!ria 

    • Histor and phsical e$amination

    • 3a#orator e)al!ation

     – Urine sodi!m

     – Urine osmolalit or specific gra)it

     – >U& creatinine

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    SHK 22

    )al!ation of *lig!ria)al!ation of *lig!ria

    3a#orator 6est -rerenal A6

    >lood Urea itrogen? C29 89:29

      (reatinine /atio

    Urine Specific ra)it C8.929 @8.989

    Urine *smolalit