soal mcq blok 10

7/29/2019 Soal MCQ Blok 10

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FrenZzz yang baik hati, soal ini jangan di fotokopi di lingkungan FK, pokoknya usahakan jangan sampaiterlihat oleh dosen, staf FK, apalagi UPEP-ers, key! Semangat y0w Belajar! Chaiy0o!!! n_n

Soal MCQ Blok 10 PDU 07

1. Bicuspid valvea. left side heart

b. Guard auricle heartc. Guard entrance aorta

d. Guard entrance pulmonalis

2. Freshly oxygenated blood enters the heart through _____ and pumped out the ___

left atrium, aorta.

3. Characteristic of ducting elastic artery (conducting artery)a. Tunica adventitia is composed by loose connective tissue

b. Vasa vasorum is subendothelial layerc. Tunica media fenestrated by elastic membraned. Thick internal elastica lamina (undulating)

4. General structure of Blood vessels in histological section, the arteri is round, with blood inthe lumina.

5. Struktur histologi otot jantunga. Multinucleated dan tidak bercabang

b. Inti sel terletak di tepic. Fusiform in shape, inti di tengahd. Individual cell branch and connect to cell via desmosome

6. Histology structure sel purkinjea. Branch

b. Bigger cellc. Cytoplasma contain abudentd. In subendothelium layer

7. The vascular system of human embrio developed froma. Mesenchymal cell

b. Angiogenetic clusterc. Cardogenic aread. Splancnic mesoderm layer

8. Bagian jantung primitif .... endocardial tube .... berubah menjadi epimiocardial mantle

9. A persons EKG dengan normal QRS n T, tidak ada P wave, pace makernya (lokasi)a. SA nodes

b. AV nodesc. Bundle Hissd. Purkinje

10.

11. Which one of the appropriate for stroke volumea. Amount by blood pumped by heart in a minute

b. Amount by blood pumped by Atriumc. EDV-ESVd. ESV-EDV

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12. The blood circulation of our body

Left ventrikelAorta Artery Capillary Vein Vein Cava Right atrium

13. In exercise, CO increased bya. BP

b. HRc. SVd. SV dan HR

14. Bunyi jantung pertama a. Tricuspid dan mitral valves closed

15. EDV=120, ESV=50, HR=70 Ditanya CO 4900

16. Hipertensi bisa menyebabkan hipertrofia. Aortab. Left ventrikelc. Right ventrikeld. Right atirum

17. In phisiology characteristic, the cardiac muscle difference with other muscle cells thata. Cardiac muscle has striate

b. Cardiac muscle work involuntaryc. Cardiac muscle is fatique resistanced. Cardiac muscle has pace maker

18. Muscle stimulate aksi potensial a. Sino Atrial nodes

19.

20. Gambar AV Blok 3

21.

22. Jika pengobatan pertama tidak berhasil, maka dilakukan Pacemaker permanent

23. A 58 yo man with history 8 days palpitation and takes warfarin. BP 128 mmHg. His exam is normal. ECGshows AF with rapid ventrikuler rate. Therapy, except

a. Digoxinb. propanolol

24. WPW syndrome is not characterized by which one followinga. This syndrome may occur in patient with valvular abnormalities, cardiomiopathy, or otherwise

normal heart functionb. The arrythmias is denoted by ventrikuler preexcitationc. The arrythmia is characterized by a delay of the impulse in the AV nodes regiond. The PR interval is shortened (the impulse reaches the ventrikel without going through the AV nodes)

due to the activating of an alternate, shorter pathway to the miocardium

25. Conduction Velocity is most rapid in which tissuea. Purkinje-His

b. Atrial

c. AV nodesd. Ventricel

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26. Repolarization of the miocardial cells is determined mostly by which currenta. Outgoing sodium

b. Ingoing calsiumc. Outgoing potassiumd. Ingoing chloride

27.

28.

29. Obat antiaritmia berdasarkan klasifikasi Vaughan Williams (Kelas I, II, III, IV). Di bawah ini temasuk obatantiaritmia tapi bukan termasuk klasifikasi adalah

a. Amiodaroneb. Procainamidec. Sotalold. Adenosine

30.

31. Digoxin can be administered orally for long term suppression of atrial flutter/fibrilation. Digoxin producestherapeutic effect by

a. Blocking Na+, K+ ATPase and indirectly increasing intracelluler calsiumb. Centrally increasing the activity of N. Vagusc. Blocking sodium channel on the membrane of cardiac celld. Increasing the force of cardiac contraction

32.

33. Efek samping amiodarone fibrose pulmonal

34. Obat antiarythmia, -blocker sotalol

35. Obat CCB punya fungsi antiarithmia, antihipertensi, dan antianginaa. Verapamil

b. Disopyramidec. Ibutilided. Quinidine

36. Class I drugs have antiarythmic action, while is resulting from the blockade of sodium channel on thecardiac cell membrane. Choose one from the listed drugs below, when is an example of class I

antiarrythmic drugsa. Propafenoneb. Diltiazemc. Propanolold. Amiodarone

37.

38. In general, treatment of asymptomatic/ minimally symptomatic arythmias should be avoided coz ...

39. Nitrogliserin adalah obat pilihan untuk serangan angina acute, efeknya adalaha. Venodilatation

b. Arteriodilatationc. Veno & arteridilatationd. Veno & arteridilatation, coronary collateral dilatasi

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40. Nitrogliserin dikonsumsi secara sublingual karena ?

41. Verapamil is a very powerful Ca antagonist that reduce the frquency and force of cardiac contraction. Bcoz

of that, verapamil should not be combinate with Propanolol

42. Beta blocker are very effective in the prophylaxis of anginal attack. The therapeutic effect are result froma. Vasodilatation

b. Decrease heart rate and force of contraction

c. Arterial vasodilatationd. Increased miocardial O2 consumption

43. Aspirin sebagai obat anti platelet bekerja dengan menghambata. Protein kinaseb. COX-1c. Cytokrom p-450d. Miosin kinase

44. Heparin is oftenly used in the treatment of acute coronary syndrome, Heparin always administeredParenterelly cause?

a. Heparin is not absorbed per oralb. Heparin is destroyed in the stripc. Heparin undergoes massive effect first pastd. Heparin can cause internal bleeding

45. Meta analisis uji klinik aspirin dan obat anti platelet lainnya confirm benefit intervensi prevention sekunderof vascular event among of the patient with history vascular event. Obat anti platelet lainnya termasuk

a. Clopidogrel & Triclodipineb. Heparinc. Warfarind. Vit K

46. Warfarin produce its anticoagulant effect by inhibit action ofa. Cyclooxigenase 2

b. Vit K reductasec. Protein kinase Ad. ACE

47. The most common morphological disorder involving valve caused by endocarditis in RHDa. Aortic insufficiency

b. Aortic stenosis

c. Mitral stenosisd. Trcuspid insufficiency

48. Pathologic disorder of the cor in RHDa. Endocarditis

b. Miocarditisc. Pancarditisd. Pericarditis

49. Microscopically features of miokarditis in RHD are found spesific granulomatous lession dispressed inmiometrium layer especially in intraventrikuler septum, the lession called

a. Veruccaeb. Mc callumc. Aschoffd. Tubercle

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50. The most underlying disease in bacterial endocarditis isa. ASD

b. ToFc. Mitral stenosis from RHDd. Pulmonary stenosis

51. The basic reaction of autoimmune process in rheumatic fever

a. Delayed reaction hipersensitivityb. Complement mediated citotoxicityc. Cross reaction antibody in tissue antigen (Cardiac antigen)d. Reaction of hipersensitivity

52. The disorder of arterioles which is thickening the wall and plugs of hyaline mass within are found ina. Chronic mild Hypertension

b. Malignant Hypertensionc. Hyperlipidemiad. Acute severe Hypertension

53. If in the microscopically finding there are a lot of foam cell in subintima layer is calleda. Atherosclerosisb. Fatty streaksc. Fatty plaqued. Injured intima

54. The mayor function of VLDL isa. Delivery cholesterol

b. Delivery of dietary fat and other fatty acidc. Delivery of carbohidrat and proteind. Picking up (scavenging) excess cholesterol from cell to liver

55. The mayor function of HDL isa. Delivery cholesterol

b. Delivery of dietary fat and other fatty acidc. Delivery of carbohidrat and proteind. Picking up (scavenging) excess cholesterol from cell to liver

56. The mayor function of cylomicron is toa. Deliver fatty acid via lipoprotein lipase

b. Deliver cholesterol from liver to cellc. Endogenous triglicerided. Membawa cholesterol dari cell ke liver

57. Chilomicrons are synthesized ina. Liver

b. Adipose cellc. Colonid. Small intestine

58. VLDL are synthesized ina. Muscle

b. Adipose cellc. Small intestined. Liver

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59. LDL are synthesizeda. From HDL

b. From IDLc. In liverd. In fat cell

60. In dislipidemia, concentration of lipoprotein

a. HDL meningkatb. LDL > 135c. Total cholesterol dan TGs < 150d. VLDL normal

61. Characteristic dislipidemia TG, HDL

62. Initial Blood preesurea. Genetic 50%

b. Environment 30 %c. One locus

d. Interact with other genes

63. Dilated Cardimyopathy (Tentang genetic disorder)a. 25 % genetik

b. Usually X-linkedc. Rare autosomal dominantd. 8 gene loci unknown

64. Faktor risiko atherosclerosis, kecualia. Male sex

b. advance agec. Hipotensid. Insulin resistance

65. Nutrition intervention of Heart failureb. Sodium 2000 mg recommendation

66. At 1st food/fase awal recommendation for ischemic heart diseasea. liquid

b. semi solidc. Soft

d. Solid

67. ECG for ASD RAD, RVH, RBBB

68.

69. What is natural history of small VSD?a. Polycitemia

b. Spontaneous closurec. Cardiomiopathyd. CHF

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70. Yang menyebabkan sianosis pada TGAa. Mixing

b. Independent pulmonary-systemic flowc. Inadequate pulmonary flowd. Increased extreme pulmo

71.

72. The phisiology of tricuspid atresiaa. All systemic venous blood is directed to LA

b. Systemic and pulmonary circulation function in parallelc. The direction a magnitude of flow through the defect depend on pulmonal stenosisd. The pulmonary and systemic circuit may be equal

73.

74. ECG pada ToF RVH

75. In long standing hypertension with normal LV function, physiological respon the heart isa. Adaptive dilatasi

b. Adaptive hypertrophyc. Dilatasi diikuti hipertrofid. All are true

76. Cause of HFa. CAD

b. Hypertensionc. Valve diseased. All above

77. Which of the following is a compensation mechanism on HFa. neurohormonal system

b. HRc. Na and water retentiond. All are true

78.

79. The true treatment of heart failure

a. Diuretics to reduce Na and water retentionb. Digitalis when there is the problem of contractilityc. B-blocker with caution for contraindication

d. All are true true

80. The following are sign of right ventricular failure, excepta. Rales

b. Hepatomegalyc. Increase JVPd. Ascites dan oedems

81.

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82. What is common associated risk factor for CADa. Dislipidemia

b. Sedentary of lifec. Cigarette smokingd. All above are true

83. Imbalance O2 supply and demand to the miocardium IHD

84. Karakteristik dari infark miokard, kecualia. Chest pain more than 15 minute

b. At times be precipitated by exertionc. Chest pain relieve after taking restd. In right coronary artery stenosis, epigastric discomfort and syncope is one of the most symptoms.

85. All the following statements can be a cause of ischemic heart disease, excepta. Spasme

b. Atherosclerosisc. Plaque ruptur

d. High HDL level

86. Hemodinamik exam having a meaning to diagnose RHDa. Hemoglobin level

b. Number RBCc. Number WBCd. LED

87. To determine etiology carditis (endocarditis and miocarditis) require inspectiona. Hematology

b. Clinical chemistryc. Urinalysisd. Blood culture

88. Which one is not including triple cardiac markera. CK MB

b. Mioglobinc. Troponind. LDH

89. Marker RHD severity

a. ASTOb. CPRc. BSRd. WBC

90. The signs of the cardiac tamponade area. Chest pain increased of the CVP, heart sound decrease

b. Hipotensi, HS decrease, fracture of the sternal bonec. JVP , HS decrease, hipotensid. Fracture of the ribs, hipotensi, normal HS

91.

92.

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93.

94. Aritmia mungkin disebabkan ketidaknormalan struktur jantungMitral Stenosis

95. Karakteristik supraventrikuler tachycardi adalah ...

96. Fact about ventrikel aritmiaa. Impulse originating from an area proximal to the AV junction

b. Have preceding corresponding P wave and the QRS compleksc. Ventrikuler takikardi is a fast, irregular beating of the heartd. The extend of the ventrikuler takikardi determine the course and necessity of treatment.

97. The right position that used for cor

PA, left lateral, LAO,RAO.

98.

99. Double contour sign of enlargement / hipertrofia. Right ventrikel

b. Left Atriumc. Right atriumd. Left ventrikel

100. Pemeriksaan jantung, kecualia. Ventriculographyb. Plain radiografi photoc. Heart Fluoroscopyd. Echocardiography

101. CTR : perbandingan cardio dan thorax

102.

103. Thoracix radiographic photo (PA) are done witha. Standing position

b. Film on behindc. Supine positiond. X-ray on front

104. Picture of enlargement of left vntrikel apex downward

105. 52 thn, , decrease visual acquify di kedua matanya. Hasil funduskopi : Internal bleeding, edema retina,hard exudately, vena dilatasi, dan berdarah. Diagnosisnya

a. Central serous retinopathyb. Age related macular degeneration (ARMD)c. Retinal detachmentd. Diabetic retinopathy

106. A 75 years old man complain blurred vission in his right eye. In fundus photograph showed surprahaemorrhages in right eye. The diagnosis

a. Retinal detachmnet

b. CRVOc. BRVOd. Hypertensive retinopathy

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107.

108. Fundusphotograph pada CRAO

109.

110. Fundus fotografi ditemukan pada pasien dengan malignan hipertrofia. Optic atrofi

b. Optic swellerc. Retinal bleeding

111. True for mild and moderate hipertensia. Retinal bleeding

b. Arteriolar narrowingc. Retinal oedemad. Microaneurism

112.

113. Less sportif in RHDa. Carditis

b. Migratory Poliarthritisc. NAd. Subacute nodule

114.

115.

116. The most common etiology of myocarditis

a. Fungaeb. Bacterialc. Virald. Tropozoa

117. Myxoma locateda. LVb. LAc. RVd. RA

118. The characteristic lesion of infective endocarditis isa. Inflammationb. Vegetationc. Destructive of valved. Differentiation of valve

119. Acute pericarditis caused by as follows, excepta. Microbacteria

b. Neoplastic diseasec. Degeneration

d. Tropozoa

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120. Infeksi dari mikrobakteri pada permukaan endothel jantung disebuta. Pericarditis

b. Miocarditisc. Infeksi endokarditisd. RHD

soal yang tercatat = 97 dari 120 soal.

eNBe : 4m all 2 all1. Sistem pencatatan, NO ABSEN & + 130.

2. Yang tidak mendapat bagian mencatat soal (soal kurang dari 130), catat inti-inti skenario. Misal jumlahsoal 120, absen no 121 mencatat skenario pertama dst., 1 skenario boleh dicatat 2-3 orang. Kerja samaantara kalian sangat diharapkan. Kompak2lah y0w!

3. Yang udah tau info ini, kasih tau ke teman lain yang belum tau, key!

4. Pilihan gandanya (a,b,c,d,e) dicatat juga, disitulah qt bisa belajar lebih. Lagipula untuk tipe multiple

choice question (soal pilihan ganda) pilihan jawabannya terkadang sedikit mengecoh.

5. Kalau bisa tetap dalam versi inggris, kadang ada yang sedikit salah mengartikan soal. Tulisannya juga!!g mesti bagus2 bener yang penting terbaca.

6. Seandainya tahu jawabannya langsung dijawab (klo bisa cari tahu jawabannya), tapi klo g yakin gdijawab juga gpp. SERSAN,serius tapi santai., Santai!! n_n thx 4 all

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soal mcq blok 10

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