dr erwin sukandi, sppd, k-kv belum

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    Dr. Erwin Sukandi, SpPD, K-KV,FINASIM

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    Nama: Tn/Ny. Tanggal: ../../.... RSMH PalembangUmur : tahun Pukul :

    Interpretasi:

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    IramaAksisHRGel. PInterval PRGel. Q, R, SInterval QRSInterval QT

    Segmen STGel. TGel. U

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    1500

    RR (Ktk kecil)atau

    300

    RR (ktk sdg)

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    Common- 2.2 million AmericansPrevalence increases with ageIncreased mortality secondary to

    stroke 75,000 strokes/year in USClassifications

    First episode

    Recurrent (paroxysmal) Persistent

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    Causes

    Advancing age

    Uncontrolled htn

    CAD CHF

    Valvular heartdisease

    Acute pulmonary

    process (PE) Hyperthyroidism Acute alcohol

    intoxication Illicit narcotic abuse

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    Absence of P wavesIRREGULARLY IRREGULARAtrial rates 350-750Ventricular rate varies

    Controlled versus rapid responseUp to 30% decrease in cardiac output

    No atrial kick

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    Treatment Goals: rate control and anticoagulation

    Rate control

    Beta-blockers and Ca channel blockers Metoprolol (Lopressor)5mg IV q 5 min times 3 doses, then oral load Diltiazem (Cardizem)10 to 20 mg SLOW IV push

    Digoxin may be used in chronic setting

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    Regular atrial activitySawtooth pattern

    Single irritable foci in atriainitiates impulse

    Atrial rate 250-300 Slower than in a-fib

    Causes: No underlying cardiac disease CAD, Htn, MI, hypoxia, digitalis toxicity,

    CHF, PE, COPD, thyrotoxicosis, alcohol

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    Treatment Ventricular rate controlBeta Blockers or Ca channel blockers

    ER/admissionAtrial overdrive pacing

    Ablation

    Adenosine may be helpful indiagnosisTransiently blocks AV node

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    Ventricular FibrillationMarquette Electronics Copyright 1996

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    Ventricular AsystoleMarquette Electronics Copyright 1996

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    Rhythm strip recording of ECG

    revealing that paroxysmal attacksof atrial fibrillation alwaysterminated with a long ventricularasystole

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    ABCs

    HIGH QUALITY CPR

    Secure the airway, provide 02

    IV or IO access

    Epinephrine 1 mg*(Repeat every 3 5 minutes)

    If the rhythm is Bradycardia,

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    The 5 Hs and the 5 Ts, while beginning drug therapyHypoxia Toxins/overdoseHypovolemia Thromboemboli (coronary/pulmonary)Hyper/hypokalemia TraumaHypothermia Tension pneumothoraxHydrogen ion/acidosis Tamponade (cardiac)

    Hypoglycemia

    Consider fluid bolus, 200-300cc; may repeat if lungs remain clear

    Once perfusing rhythm is restored, maintain adequate ventilations,and then stabilize

    Rate, Rhythm, and Blood Pressure

    Note: Repeated unsuccessful intubation attempts are notrecommended. BVM support of the airway is acceptable until

    advanced airway can be placed.*You may consider Vasopressin instead of the first or second dose

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    ABCs, EFFECTIVE CPR

    SECURE AIRWAY

    IV/IO access

    Epinephrine 1.0 mgOR

    Vasopressin 40u(one time only instead of 1st or 2nd epi dose)

    Atropine 1 mg

    Epinephrine 1 mg

    Atropine 1 mg

    Epinephrine 1 mg

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    Several factors should be considered when making the decision to terminate

    resuscitation efforts on a patient in extended Asystole:Down Time Rigor MortisCold Water Drowning Chronic Medical Conditions

    Age Skin TemperatureBlood Pooling Trauma

    And most importantly.quality of life!

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    ABCs

    Start HARD, FAST, EFFECTIVE CPRIf un-witnessed code or down time > 4 minutes,

    2 minutes of CPRprior to defibrillation

    Defibrillate 200 j biphasic(or device specific dose, 360j if monophasic)

    Continue CPRimmediately w/o pulse rhythm check

    Secure the airway without prolonged intubation attempts

    And establish IV or IO with Saline or LR

    Vasopressin 40 Units I.V.DURING CPR

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    OREpinephrine 1 mg I.V.

    (Repeat every 3 5 minutes)

    Defibrillate200-360 joules (Repeat every 1 2 minutes)

    Amiodarone 300 mg I.V.OR

    Lidocaine 1 - 1.5 mg/kg I.V.(May repeat to a max of 3 mg/kg)

    Defibrillate200-360 joules

    Consider Sodium Bicarbonate 1meq/kg(acidosis, tricyclic overdose, or hyperkalemia)Consider Magnesium Sulfate 1 2 grams I.V. (if Torsades is present)Upon return of spontaneous circulation (ROSC): Assess airway, breathing andvital signs. Provide medication appropriate for heart rate, rhythm and bloodpressure. Consider hanging a maintenance anti-arrhythmic drip upon ROSC

    to prevent reoccurrence of V-Fib.

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    Assess ABCs

    Secure airway, provide oxygen, pulse oximetry

    Start IV

    Revised 9/2009 10Obtain 12 lead EKG if possible, and review patients history,

    especially history of possible A-fib or A-flutter

    If determined a new onset, consider synchronized cardioversion@

    100, 200, 300, 360 joules(Consider Sedation)

    Cardizem 0.25 mg/kgOR

    Verapamil 2.5 5 mg

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    *Note: never delay cardioversion in lieu of sedation if the patientis unstable. (You can always apologize later)

    If AF has been present for >48 hours, a risk of systemic

    embolization exists with conversion to sinus rhythm unlesspatients are adequately anticoagulated for at least 3 weeks.

    Electrical cardioversion and the use of antiarrhythmic agentsshould be avoided unless the patient is unstable orhemodynamically compromised.

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    Which one is more tachycardicduring this exercise test?

    TERIMA