klasifikasi berdasarkan workshop american academy of periodontology

8/10/2019 Klasifikasi Berdasarkan Workshop American Academy of Periodontology

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KLASIFIKASI BERDASARKAN WORKSHOP

AMERICAN ACADEMY OF PERIODONTOLOGY

(AAP) 1999

I. GINGIVAL DISEASES

A. Dental plaque-induce gingival disease

I. Gingivitis associated with dental plaque only

a. without other local contributing factors

b. with local contributing factors

2. Gingival disease modified by sistemic factorsa. associated with the endocrine system

1) puberty associated gingivitis

2) menstrual cycle associated gingivitis

3) pregnancy associated

a) gingivitis

b) pyogenic-granuloma

4) diabetes melitus associated gingivitisb. associated with blood dyscrasias

1) leukemia associated gingivitis

2) other

3. Gingival diseases modified by medications

a. drug-induced gingival disease

1) drug induced gingival enlargement

2) drug induced gingivitis

a) oral contrasepsis associated gingivitis

b) other

4. Gingival diseases modified by malnutrition

a. ascorbic acid deficiency gingivitis

b. other

B. Non-plaque induced gingival diseases

1. Gingival diseases of spesific bacterial origin


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a. Neisseria gonorrhea associated lesion

b. Treponema pallidum associated lesion

c. Streptococcal spesies associated lesion

d. other2. Gingival diseases ofviral origin

a. herpes virus infections

1) primary herpetic gingivostomatitis

2) recurrent oral herpes

3)varicella zoster infections

b. other

3. Gingival diseases of fungalorigin

a. candida spesies infection

1) generalized gingival candidosis

b. linear gingival erythema

c. histoplasmosis

d. other

4. Gingival lesions of geneticorigin

a. hereditary gingival fibromatosis

b. other

5. Gingival manifestation of systemic condition

a. mucocutaneous disorders

1) lichen planus

2) pemphigoid

3) pemphigus vulgaris4) erythema multiforme

5) lupus erythrmatosus

6) drug-induced

7) other

b. allergic reactions

1) dental restoratif materials

a) mercury


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b) nickel

c) acrylic

d) other

2) reactions attributable toa) tooth paste/ dentifrices

b) mouthrinses/mouth washes

c) chewing gum additives

d) foods and additives

3) other

6. Traumatic lesions ( factitious, iatrogenic, accidental)

a. chemical injury

b. physical injury

c. thermal injury

7. Foreign body reactions

8. Not otherwise specified

II. CHRONIC PERIODONTITIS

** prevalent in adult but can occur in children

** amount of destruction consistent with local factor

** associated with a variable microbial pattern

** subgingival calculus frequently found

** slow to moderate rate of progresion with possible periods of

rapidprogresion

** possibly modified by or associated with the following

systemic diseases : DM, HIVlocal factor predisposing periodontitis

enviromental factor: smoking, emotional stress

A. Localized----- < 30% sites involved

B. Generalized---- > 30% of sites involved

* Slight : 1 to 2 mm of clinical attachment loss

*moderate: 3 to 4 mm

*severe: > 5 mm


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III. AGGRESSIVE PERIODONTITIS

* Otherwise clinically healthy patien

Rapid attachment loss and bone destruction

Amount of microbial deposits inconsistent with diseasesseverity

Familial aggregation of diseases individuals

A.Localized

* circumpubertal onset diseases

localized first molar or incisor diseases with proximal

attachment loss on at least 2 permanent teeth, one of which

is a first molar

* robust serum antibody response to infecting agents

B. Generalized

usually affecting person under 30 years of age (however

may be older)

generalized proximal attachment loss affecting at least 3

teeth other than first molar and incisor

* pronounced episodic nature of periodontal destruction

* poor serum antibody response of infecting agents

IV . Periodontitis as Manifestationof Systemic diseases

A.Associated with hematological disorders

1. Acquired neutropenia

2. Leukemias

3. otherB. Associated with genetic disorders

1. familial and cyclic neutripenia

2. down syndrome

3. leukocyte adhesion deficiency syndromes

4. papilon lefevre syndrom

5. chediak higashi syndrome

6. histiocytosis syndrome


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7. glycogene storage diseases

8. infantile genetic agranulocytosis

9.cohen syndrom

10. ehlers danlos syndrom11. hypophosphatasia

12. other

C. Not otherwise specified (NOS)

V. Necrotizing Periodontal diseases

A.Necrotizing Ulcerative Gingivitis

B.Necrotizing Ulceratif Periodontitis

VI. Abcesses on Periodontium

A.Gingival abces

B.Periodontal abces

C.Pericoronal abces

VII. Periodontitis associated with endodontic lesions

A. Combined periodontic endodontic lesion

VIII. Developmental or Acquired deformities and conditions

A.Localized tooth related factor that modify or predispose to

plaque induced gingival diseases/periodontitis

1. tooth anatomic factor

2. dental restoration/ appliances

3. root fractures

4. cervical root resorption and cemental tears

B. Mucogingival deformities and condition around teeth1. Gingiva/soft tissue resesion

a. facial or lingual surface

b. interproximal/ pappllary

2. lack of keratinized gingiva

3. decreased vestibular depth

4. Aberrant frenum/ muscle position

5. Gingival excess


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a. pseudo pocket

b. inconsistent gingival margin

c. excessive gingival display

d. gingival enlargement6. Abnormal color

C. Mucogingival deformities and condition on edentulous

ridge

1. vertical/horizontal ridge deficiency

2. lack of gingiva/ keratinized tissue

3. gingival/soft tissue enlargement

4. Aberrant frenum/muscle position

5. Decreased vestibular depth

6. abnormal color

D. Occlusal trauma

1. primay

2. secondary

klasifikasi berdasarkan workshop american academy of periodontology

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