interpretasi data klinik gangguan ginjal

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Oleh : Ritha widya S.Si,MARS,Apt PROGR AM STUDI PROFESI APOTEKER FAKULTAS FARMASI UNIVERSITAS ! AGUSTUS "#$ %AKARTA

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Page 1: Interpretasi Data Klinik Gangguan Ginjal

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Oleh :Ritha widya S.Si,MARS,Apt

PROGRAM STUDI PROFESI APOTEKERFAKULTAS FARMASI

UNIVERSITAS ! AGUSTUS "#$ %AKARTA

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LATAR&ELAKANGKe'(t(ha) *e+a)- Ap+tee aa) pe/aha/a)/e)-e)ai data li)i ()t( dapat /ela(a)

ti)daa) e0a/a*ia) de)-a) tepat *e*(aide)-a) t(1(a) teapi

&UKAN UNTUK MENDIAGNOSA

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 Ti)1a(a) data li)i -i)1alO-a)+lepti* (i) : 2a)a da) &a( (i)

Pe/ei*aa) i/ia (i): Gl(+*a, A*et+),

&ili('i), Daah,&eat %e)i*, P+tei),p3Mi+*+pi (i) : *ili)de da) *el

Ki/ia daah : eati)i), &UN

Pe)ilaia) 0()-*i -i)1al : 4lta*i da) t('(l(*

 Ti)1a(a) pe/ei*aa) adi+l+-i

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ORGANOLEPTIS URIN

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2ARNA URINN+/al : 1e)ih *ediit 'ea'(t da) 'ewa)a

()i)-,

i)te)*ita* wa)a li)ie de)-a) pe)i)-ata)+)*e)ta*i

Kee(ha) : i*tali*a*i (at50+*0at

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2ARNA URIN

Meah

Pat+l+-i : 3ae/+-l+'i)

N+) pat+l+-i : +)*(/*i +'at, 6at wa)aOa)ye

Pat+l+-i : Pi-/e) e/ped(, 'ili('i)

N+) pat+l+-i : pe)--()aa) +'at ISK, piidi(/

K()i)-

Pat+l+-i : 'ili('i), (+'ili('i), (i) peat

N+) pat+l+-i: w+tel, )it+0(a)ti+)

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2ARNA URIN 78+)td..93i1a(

Pat+l+-i : 'iliedi), 'atei 7p*e(d+/+)a*9

)+) Pat+l+-i : ita/i), +'at p*i+aati0, di(e*i*

&i()+) pat+l+-i : di(eti, )it+0(a)

8+lat

pat+l+-i : pi-/e) e/ped(

)+) pat+l+-i : le+d+pa, )it+0(a), *(l0a3ita/5hita/ e;+lata)

Pat+l+-i : (+'ili)+-e)

)+) pat+l+-i : +/ple* 'e*i, 0e)+l

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&AU URIN

N+/al : *ediit 'e'a(

&a( a*et+)  pada dia'ete* et+a*id+*i*,

&a( a/+)ia  i)0e*i 'atei 7(ea a/+)ia9

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KIMIA URIN

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PEMERIKSAAN KIMIA

URINGl(+*a   N : )e-ati0 

A*et+)  N : )e-ati0 

&ili('i)  N: )e-ati0 

Daah  N : )e-ati0 

P+tei)

&eat %e)i*p3

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PROTEINURIA

Ke'eadaa) p+tei) di dala/ (i)

P+tei) (/(/)ya /e(paa) *e)yawade)-a) &M 'e*a, *ehi)--a pada eadaa))+/al *(lit dapat te4lta*i +leh -l(/e(l(*

P+tei) apa'ila te4lta*i /aa aa)teea'*+'*i e/'ali e *i(la*i *i*te/i

U1i dip*ti : < = ># 7># /e)()1(a)+)*e)ta*i p+tei) ya)- ti)--i9

U1i la' : (a)titati0 de)-a) i)*t(/e)

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KENAPA &ISA TER%ADI

PROTEINURI ???

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MEKANISME

PROTEINURIA• F()-*i+)al

 Te1adi pada -i)1al )+/al, ae)a : +lah a-a

'eat, de/a/, pe)i)-ata) ee*i p+tei)ae)a 'edii la/a

• Alia) pa e)al

Pe)i)-ata) ada p+tei) 'e &M e)dah ya)-

dii(ti de)-a) pe)i)-ata) 'e'a) 4lta*i-l(/e(l(*   ea'*+'*i p+tei)

 

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MEKANISMEPROTEIN....;+)tdGl(/e(l(*

&eaita) de)-a) pe)yait -l(/e(l(* e@.

-l(/e(l()e0iti* T('(l(*

U/(/)ya p+tei)(ia 'eat ae)a e(*aa)t('(l(*, e@. pyel+)e0iti* da) )e+*i* t('(l(*

a(t.

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KLASIFIKASI

PROTEINURIAP+tei) <,$ -5h

OR 'eat, *te**, hipet)*i dll

P+tei) *eda)- <,$=B -5 haiGN +)i*, )e0+pati dia'ete*, pe ela/*ia

P+tei) &eat C B -5hai

GN a(t, GN +)i* 'eat, )e0+pati 'eat

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p3 URIN

p3 (i) dewa*a )+/al : $ = ,$ , ata :

p3 (i) a*a/ :

Ke(*aa) 1ai)-a), diet hewa)i, a*id+*i*/eta'+li5e*piat+i, de/a/

p3 (i) 'a*a :

Diet *ay(a) '(ah, ISK

7p*e(d+/+)a*5p+te(*9, *(*(Pe)-a(h a*(pa) tehadap ea*a/a) dapat

'e/a)0aat dala/ teapi 'at( -i)1al

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&at( ya)- *i0at)ya a*a/ : al*i(/ +*alat,a*a/ (at5*i*tei)

&at( ya)- *i0at)ya 'a*a : al*i(/ 0+*0at5/a-)e*i(/ a/+)i(/ 0+*0at

URIN ASAM DAPAT MENGOBATI BATU GINJAL BASA

URIN BASA DAPAT MENGOBATI BATU GINJAL ASAM

Pada penderita batu ginjal!eberadaan

!"n#tituen ter#ebut didala$ urin dapat

$e$pridi!#i!an adan%a jeni# !ea#a$an batu

ginjal

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&ERAT %ENIS URINPe)-((a) de)-a) (i)+/ete ()t(

/e)e)t(a) +)*e)ta*i (i)

&1 (i):

N+/al : ,<<=,<B$Di'ei /i)(/ 'a)ya : ,<<

Ke(a)-a) ;aia) : ,<#

BJ urin perlu $e$perti$bang!an a#upan

untu! $engeta&ui !e$a$puan pe$e!atan urin

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MIKROSKOPIK URIN

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UNSUR=UNSUR A&NORMAL

URINE Sel=*el

Sile)de

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SILINDER

Ma*a p+tei) 'e'e)t(

*ili)di* ya)- te'e)t(dit('(l(* -i)1al da)

di'ila* /a*( e dala/(i)

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 %e)i*=1e)i* *ili)deSili)de di'a-i 'eda*aa) 'e)t()ya :

Sili)de hiali)  OR 'eat

Sile)de *el daah /eah

 -l(/e(l(*)e0iti*Sili)de *el daah p(tih  ISK5pyel+)e0iti*

Sili)de *el epitel t('(l(*  )e+*i* t('(l(*

Sili)de 'atei  ISK5pyel+)e0iti*

Sili)de le/a 7'ada) +al9  p+tei)(i5)e0+ti *i)d+/

Sili)de -a)(la -a-al -i)1al

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SelNe(t+4l i)0e*i

E+*i)+4l  i)0e*i

Li/0+*it  ada)ya e*i pe)+laa) padap+*e* ta)*pla)ta*i

Sel epitel  -a)--(a) t('l(*

Eit+*it  e(*aa) -l(/e(l(*

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KIMIA DARA3

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&LOOD UREA NITROGEN7&UN9Nit+-e) (ea /e(paa) p+d(

/eta'+li*/e p+tei)

&UN N+/al : < < /- 5 << /lK+)*e)ta*i te-a)t()- dai a*(pa)

Apa'ila GFR t(() te1adi pe)i)-ata) &UN

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KREATININKeati)i) /e(paa) p+d( /eta'+li*/e

+t+t

 Te4lta*i +leh -l(/e(l(*, teee*i dala/'e)t( (t(h, teea'*+'*i *e'a-ia) e;ildala/ -i)1al

Keati)i) *e(/ )+/al : <,!=,$ /-5<< /l

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PENILAIAN FUNGSIGIN%ALF()-*i 4lta*i

F()-*i t('(l(*

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GLUMERULUS FILTRATE RATE

7GFR9Ke/a/p(a) -l(/e(l(* dala/ /e/4lta*i

la(ta)

Met+de :

Medi;al A)i/ati+) Kid)ey F();ti+) i) Filte,i)- 8+)t,a*t.H

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INULIN KLIRENSSi0at :

Se)yawa P+li*aaida 0(t+*a

 Tida teiat p+tei) pla*/a, Tida te*ee*i da) tida teea'*+'*i

 Tida te/eta'+li*/e +leh -i)1al

I)(li) di'eia) /elal(i tete*a) IV +)*ta) (5

/e)1a/i) ete*ediaa) didala/ pla*/a ya)-+)*ta)

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INULIN KLIRENS

GFR = Kli in = (U in x V) / P in

U i) : +)*e)ta*i i)(li) dala/ (i) 7/-5dl9

P i) : +)*e)ta*i i)(li) dala/ pla*/a 7/-5dl9

V : V+l(/e (i) 7 /l5/e)it9

Kl i) : lie) i)(li) 7 /l5/e)it9

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KREATININ KLIRENS8+;+0t da) Ga(lt :

  Pia : Kl J 7#<=(/(9 @ && 5 7! S 9

  2a)ita : Kl @ <,$

 %ia +'e*ita* && di-a)ti de)-a) I&2I&2 :

Pia $< >7,B @ ele'iha) ti)--i $ 0eet dl/ i);hi9

2)t #$,$ > 7,B @ ele'iha) ti)--i $ 0eet dl/ i);hi9

'et ( 'l !r ( !reatini !liren# )$l*$enit+

U$ur ( ta&un

IB, ( ideal b"d% -eig&t )!g+

Sr !r ( #eru$ !reatinin

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E*ti/ate Re)al F();ti+)Modifed Diet in Renal Disease (MDRD)

GFR (mL/min/1.73 m ) = 1!" x #$%& '

1.1 x (*+e) ' ,.,3 x (,.7 i- -emale)x (1.1, i- *-%ian ' *me%ian)

S8: *e(/ ;eati)i)e i) /-5dL a-e i) yea*

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*+alI'( *+/ad (/( B tah(), 'eat 'ada) #<

- , /e)-el(h )yei (i)a*i, *etelah di ;eeati)i) *e(/ )ya *e'e*a : <,# /-5dl.

&eapaah lie)* i'( *+/ad ?

8;l J7#<=B9 @ #< @ <,$

  ! @ <,#

<!,B /l5/e)it

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GFRStage  Description GFR 

ml/min/1.73m² 

Action*

1 Kidney damage witnormal or !GFR

" #$.$ Diagnosis ant treatment%

&reatment o' comor(idconditions.

Slowing progression.

)D ris+ red,ction 

- Kidney damage witmild GFR

$0#2stimating progression 

3 oderate GFR 3$04# 25al,ating and treatingcomplications

6 Se5ere GFR 140-# reparation 'or +idneyreplacement terapy

4 Kidney 'ail,re

8 ESRD9:14 8 or dialysis9  Replacement 8 i'

,remia present

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 TEST FUNGSI TU&ULUSU)t( /e)-etah(i 0()-*i t('(l(*

Met+de :

 Te* PSP Te* Paa a/i)+h(tiat 7PA39

 Te* pe/eata) da) pe)-e);ea)

 Te* pe)-a*a/a) (i)

 Te* +)*ea*i )ati(/

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NILAI NORMAL URINALISALe(+*it : <=# 5 LP&

Sel epitel : <=$ 5LP&

&atei : <

&ada) le/a +al : <Sili)de : <= 5 LP& 7hiali)9

Gl(+*a : )e-ati0 

Ket+) : )e-ati0 

Eit+*it : <= 5 LP&Le(+*it : <=# 5 LP&

Spe/at+6+a : > 7lai=lai9

Le)di

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PEMERIKSAAN

RADIOLOGI

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Piel+-a/ I)tae)a 7IVP9

U)t( /elihat elai)a), *epeti : at+4 -i)1al,piel+)e0iti*, da) -a/'aa) p+*i*i *etae'eadaa) -i)1al

Met+de : 0+t+ p+l+* adi+-a4 a'd+/e)  pe)y()tia) /edia +)ta* di*()tia) *e;aa dila(a) pe/+teta) tiap /e)it pada $/e)it peta/a

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Ulta*+)+-a4 Gi)1al

Me/'edaa) t(/+ padat de)-a) i*ta ya)-/e)-a)d()- ;aia)

U(a) -i)1al

Ada)ya +'*t(*i

Ada)ya pe)+laa) -i)1al pada pe);a)-+a)

Me)e)t(a) leta -i)1al pada p+*e* 'i+p*i

-i)1al

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Radi+)(lida 7i*+t+pi9

Me)--()aa) pe)y()tia) adi+ati0 ()t(/elihat p+*e*=p+*e -i)1al 7 4lta*i, *ee*i,ee*i, -a)--(a) di0(*i 9

&ia*a di-()aa) ()t( /elihat e0eti4ta*e1a -i)1al *etelah pe);a)-+a)

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8T= S;a)

Me/pelihata) -a/'aa) /eli)ta)-

Me)--a/'aa) *e;aa teliti *el((h *i*te/(i)ai(*

Pe)etapa) *tadi(/ )e+pla*/a

Dapat /e/pelihata) 'at( pada ta;(*(i)a(*

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MRI&e0()-*i *a/a *epeti 8T=*;a), )a/()/et+de i)i tida /e/'(t(ha) pa1a)a)adia*i i+)

Me/'eia) -a/'aa) le'ih i);i di'a)di)-8T=*;a)

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&IOPSI GIN%ALU)t( /e)dapata) *pe*i/e) 1ai-a) -i)1al,ya)- e/(dia) *pe*i/e) dipeia* de)-a)/i+*+p ;ahaya, elet+) da)

i/()+H(e*e)

8(plia) dia/'il /elal(i 1a(/ ya)-di*()tia) eaah -i)1al de)-a) /et+deh(*(* da) dila(a) +leh ahli )e0+l+-i

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 TERIMA KASI3