Download - BATU SALURAN KEMIH
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BATU SALURAN KEMIH
Syaiful AzmiSub Bagian Ginjal Hipertensi Bagian Penyakit Dalam
Fakultas Kedokteran Universitas Andalas/RSUP Dr.M.Djamil Padang
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SEJARAH DAN EPIDEMIOLOGI
Mesir 1901Batu pada mummi zaman 7000- 3000 SMInsiden ↑As : 5,2%Umur : Dekade 3-5Jender : Laki-lakiKambuh : 50%
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Lokasi
• Ginjal : Nefrolitiasis• Ureter : Ureterolitiasis• Kandung Kemih : Visikolitiasis• Uretra : Uretrolitiasis
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komposisi Kalsium
• Oksalat• Fosfat
Asam urat Struvit Sistin Lain- lain
• Xantin• Indigo
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Faktor RisikoIntrinsik o Riwayat keluargao Umuro Jender
Ekstrinsik o Diet (Protein hewani)o Intake cairano Obeseo Hipertensio Gouto Hiperparatiroido Dll
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Patogenesis• Interaksi
– Faktor Genetik– Faktor Biologik
• Faktor Biologik– Supersaturasi– Ph Urin– Faktor Protektif
• Supersaturasi– Konsentrasi bahan tertentu melebihi daya larut dalam urine– Misal :
» As oxalat» As urat» Sistin» xantin
• Faktor Protektif– Sitrat– Magnesium– pirofosfat
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Batu Kalsium• 70 %• Faktor Risiko
• Hiper kalsiuria• Hiper urikosuria• Hiper oxaluria• Hipo sitraturia
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Batu AS Urat
• Faktor Risiko • Hiper urikosuria• Ph urin asam
• Radiolusen
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Diagnosis AN :
– Nyeri (kolik)– Hematuri (mikro/makro)
PE :– Bila sudah ada komplikasi– Ureterolitiasis : Hidronefrosis– Uretrolitiasis : Retensiourine– Infeksi– PGK
LAB : – Urinalisis :
• Protein uria• Hematuria• Lekosituria
– Radiologi :• BNO /IVP• USG• CT Scan
– Analisis Batu
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Terapi Konsertatif :– Analgetik – Batu ≤ 5mm – Urin yang cukup 68% keluar– Obat-obat :
• Ca Chanel Blocker• Spasmolitik• Α Bloker
Bedah– ESWL– URS– PCNL– Open
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Pencegahan • Intake cairan out put 2-3 L/Hr• Diet rendah protein : protein hewani• Batu urat : rendah purin• Batu kalsium : rendah kalsium• Batu oksalat : rendah oksalat, coklat, kacang
Prognosis • baik bila tidak ada komplikasi
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Terima kasih