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Cedera Kepala KULIAH CMS 2013: DR. dr. Robert Sinurat, SpBS (K)

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Page 1: CMS 2014

Cedera Kepala

KULIAH CMS 2013:DR. dr. Robert Sinurat, SpBS (K)

Page 2: CMS 2014

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InsidensiUSA:

- 200/ 100.000 penduduk (600.000 kasus per tahun)

- Populasi tertinggi usia 15-24 tahun- Laki-laki >2 kali daripada wanita

- CKB: 10 % (Anderson, dkk)

Indonesia:Thn 2000: 1841 orang dirawat di RSHS Bandung, CKB: 144 orang (12,8 %) (R.Sinurat)

Page 3: CMS 2014

Bahan Buku TEKS:

• 1. Neurosurgery: Youmans

• 2. Head Injury: Pieter Reily

• 3. Head injury: Narayan

• 4. Ilmu Bedah Saraf: Prof. Satyanegara

• 5. Cedera Kepala: Prof. Eka W

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CPP = MABP - ICP

• CPP ≥ 70 mmHg

• MABP= S + 2D

3

• ICP = 0 – 10 mmHg

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Hukum “Monro Kellie”

Volume intra kranial :• Jaringan otak • Darah Konstan• LCS

Volume tek. intra kranial herniasiPerdarahanEdema otak

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Mekanisme Cedera Kepala

• Fleksi

• Ekstensi Akselerasi - deselerasi

• Shearing

Page 10: CMS 2014

1. Cedera primer

Primary traumatic brain damage Diffuse:

1. Diffuse Axonal Injuryal 2. Diffuse Vascular Injury

Focal : 1. Vascular injury resulting in:

- Intracerabral hemorrhage - Subdural hemorarhage - Extradural hemorrhage

(EDH)

2. Axonal injury 3. Contusion 4. Laceration

Page 11: CMS 2014

2. CEDERA SEKUNDER

Secondary brain damage

Diffuse

1. diffuse hypoxic-ischemic damage

2. diffuse brain swelling

Focal

1. Focal hypoxic-ischemic injury

2. Focal brain swelling

Page 12: CMS 2014

Penyebab Cedera Sekunder Ekstra dan Intrakranial

• Ekstrakranial- Hipoksia - Hipotensi - Hiponatremi

- Hipertermia - Hipoglikemia

• Intrakranial- Perdarahan

- Ekstradural(EDH) - Subdural- Intracerebral - Intraventikular- Subarachnoid

- Edema- Infeksi

- Meningitis

- Abses otak

Page 13: CMS 2014

Dasar Molekuler Cedera Otak

1. Radikal Bebas

2. Gangguan Homeostase Kalsium

3. Efek Cytokine

4. Mekanisme Lain: - Reseptor Opiat

- Konsentrasi Magnesium Intraselluler

Page 14: CMS 2014

Primary Traumatic Insult

Hypoxia IschemiaHypotension

↓ Energy Metabolism

Free Radical ExcitatoryRelease Cytokines Endorphines

Membrane Damage

Cell Death

Page 15: CMS 2014

GL NMDA ReceptorU Ca++ HydrolisisTAM AMPAA Na Cell SwellingT

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PENATALAKSANAAN1. Primary Survey

A. Airway, dengan kontrol servikal B. BreathingC. CirculationD. DisabilityE. Exposure

Resusitasi2. Tambahan terhadap primary survey3. Secondary survey4. Tambahan terhadap secondary survey5. Reevaluasi6. Terapi definitif

Page 17: CMS 2014

Disability

1. NILAI: a. Glasgow Coma Scale (GCS)

b. Periksa Pupil: Bulat, diameter?

Refleks Cahaya?

c. Motorik: Hemiparese Kiri/Kanan

Kesimpulan:

GCS 3-8 : Cedera Otak Berat

GCS 9-12: Cedera Otak Sedang

GCS 13-15: Cedera Otak Ringan

Bila ada pupil dilatasi dan hemiparese: Lateralisasi

Page 18: CMS 2014

Tindakan:

Cedera Otak Berat:

- Intubasi dan Ct scan kepala tanpa kontras.

Cedera Otak Ringan dan Sedang: Ct scan kepala tanpa kontras

Page 19: CMS 2014

Tn Rosid (32 Tahun): GCS Berapa?

VID-Tn.Rossid-ICU UKI.3GP

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?

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