1. soetomenggolo ts. kejang demam. dalam : soetomenggolo ts

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40 DAFTAR PUSTAKA 1. Soetomenggolo TS. Kejang demam. Dalam : Soetomenggolo TS, Ismael S, penyunting. Buku Ajar Neurologi Anak. Edisi ke 1. Jakarta : BP IDAI ; 1999. p. 244-51. 2. Pusponegoro HD. Kejang demam patofisiologi dan penatalaksanaannya. Dalam: Kustiowati E, ed. Kumpulan makalah pertemuan nasional I epilepsi. Semarang : Penerbit UNDIP ; 2004. p. 149-55. 3. Dadiyanto Dwi W, dkk. Buku Ajar Ilmu Kesehatan Anak. Semarang ; Badan Penerbit Universitas Diponegoro; 2011 4. Jonhston MV. Seizure in childhood and prevalence of febrile seizure. In : Behrman RE, Kliegman RM, Jenson HB, ed. Nelson Textbook of Pediatrics. 18th ed. Philadelphia : WB Saunders Co;2007.p.2457-71 5. Bessisso MS, Elsaid MF, Almula NA, Kadomi NK, Zeidan SH, Azzam SB, dkk. Recurrence risk after a first febrile convulsion. Saudi Med J 2001;22:254-8 6. Kjeldsen MJ, Corey LA, Solaas MH. Genetic factors in seizures: a population based study of 47.626 US, Norwegian and Danish twin pairs. Twin res hum genet 2005;8138-47 7. Abolfazl Mahyar, Parviz Ayazi, Mazdak Fallahi, and Amir Javadi. Risk Factors of the First Febrile Seizures in Iranian Children. Hindawi Publishing Corporation International Journal of Pediatrics 2010; 1115; 2 - 3

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Page 1: 1. Soetomenggolo TS. Kejang demam. Dalam : Soetomenggolo TS

40

DAFTAR PUSTAKA

1. Soetomenggolo TS. Kejang demam. Dalam : Soetomenggolo TS, Ismael

S, penyunting. Buku Ajar Neurologi Anak. Edisi ke – 1. Jakarta : BP IDAI

; 1999. p. 244-51.

2. Pusponegoro HD. Kejang demam patofisiologi dan penatalaksanaannya.

Dalam: Kustiowati E, ed. Kumpulan makalah pertemuan nasional – I

epilepsi. Semarang : Penerbit UNDIP ; 2004. p. 149-55.

3. Dadiyanto Dwi W, dkk. Buku Ajar Ilmu Kesehatan Anak. Semarang ;

Badan Penerbit Universitas Diponegoro; 2011

4. Jonhston MV. Seizure in childhood and prevalence of febrile seizure. In :

Behrman RE, Kliegman RM, Jenson HB, ed. Nelson Textbook of

Pediatrics. 18th ed. Philadelphia : WB Saunders Co;2007.p.2457-71

5. Bessisso MS, Elsaid MF, Almula NA, Kadomi NK, Zeidan SH, Azzam

SB, dkk. Recurrence risk after a first febrile convulsion. Saudi Med J

2001;22:254-8

6. Kjeldsen MJ, Corey LA, Solaas MH. Genetic factors in seizures: a

population based study of 47.626 US, Norwegian and Danish twin pairs.

Twin res hum genet 2005;8138-47

7. Abolfazl Mahyar, Parviz Ayazi, Mazdak Fallahi, and Amir Javadi. Risk

Factors of the First Febrile Seizures in Iranian Children. Hindawi

Publishing Corporation International Journal of Pediatrics 2010; 1115; 2 -

3

Page 2: 1. Soetomenggolo TS. Kejang demam. Dalam : Soetomenggolo TS

41

8. Parmar RC, Sahu DR. Bavdekar SB. Knowledge attitude and practices of

parents of children with febrile convulsion. J postgrad Med 2001;47:19-23

9. Pisacane A, Sansone R, Impagliazzo N, Cappolo A, Rolando P, Tregrossi

C, et al. Iron deficiency anemia and febrile convulsion. BMJ 1996 ; 12 :

313-43.

10. Kobrinsky NL, Yager JY, Cheang MS, Randall WY, Terenbein M. Does

iron deficiency raise the seizure threshold ?. J Child Neurol 1995 ; 10 :

105-9.

11. http://www.indexmundi.com/facts/indonesia/prevalence-of-anemia 7 12

2013

12. Dirjen Kesmas RI. Situasi gizi terkini dan penanggulangan masalah gizi

di Indonesia. Departemen Kesehatan RI, 2000 Juli

13. Michael J. Gibney,dkk . Gizi Kesehatan Masyarakat. Jakarta . EGC : 2008

14. Fuadi, Tjipta Bahtea, Noor W. Faktor Resiko Bangkitan Kejang Demam

Pada Anak. Sari Pediatri. 2010;12(3):142-9.

15. Kenneit F Swaiman, Stephen Ashwal, Donna M. Ferriero. Pediatric

Neuroloy Principles and Practice. Ed 4th. Phyladelphia . MOSBY : 2006

16. Fisher RS, Wu J. Basic electrophysio logy of febrile seizures. In: Baram

TZ, Shinnar S, ed., Febrile Seizures. San Diego : Academic

press;2002.p.23 1-47.

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42

17. Tath Z, Yan XX, Hafteglou S, Ribak CE, Baram TZ. Seizure-induced

neuronal injury: vulnerability to febrile seizures in an immature rat model.

J Neurosci 1998;18:4285-94.

18. Wu J, Fisher RS. Hyperthermic Spreading Depressions in the immature

Rat Hippocampal Slice. J Neurophysiol 2000; 84(3): 1355-60.

19. Jensen F E. Sanchez R M. Why Does the Developing Brain Demonstrate

Heightened Susceptibility to Febrile and Other Provoked Seizures?. In:

Baram T Z, Shinnar S. ed. Febrile Seizures. San Diego: Academic

Press;2002.p. 153- 62.

20. Berg AT. Recurrent Febrile Seizures. In: Baram FZ, Sinnar S.ed. Febrile

Seizures. San Diego: Academic Press;2002.p.37-49.

21. Menkes JH, Sankar R. Paroxysmal Disorders. Dalam:.Menkes JH, Sarnat

BH, penyunting. Child neurology. Edisi ke-6. Philadelphia: Lippincott

Williams & Wilkins JR; 2000.h.987-91.

22. Hauser WA, Anneger JE, Anderson E, Kurland LT. The risk of seizures

disorders among relatives of children with ferible convulsions. Neurology

1985;35:1268-73

23. Beard J. Iron deficiency alters brain development and functioning. J Nutr

2003 ; 133: 1468-72.

24. Mittal RD, Pandey A, Agarwal KN. Effect of latent iron deficiency on

GABA and glutamate neuroreceptors in rat brain. Indian J Clin Biochem

2002; 17 (2) : 1-6

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43

25. Guyton and Hall. Buku Ajar Fisiologi Kedokteran, edisi 11. Jakarta . EGC

: 2006

26. Nathan DG, Orkin SH, Oski FA, Ginsburg D. Nathan and Oski’s

Hematology of Infancy and Childhood. 7th ed. Philadelphia: Saunders;

2008.

27. Bambang P H, Sutaryo, Ugrasena I, Windiastutui E, Abdulsalam M. Buku

Ajar Hematologi – Onkologi Anak. Ikatan Kedokteran Anak Indonesia;

2005. P. 24 – 58.

28. Corwin. Buku Saku Patofisiologi. Jakarta : EGC; 2009

29. Jafar Nurhaedar, Peranan gizi pada anemia ibu hamil. Makassar .

Universitas Hasanudin : 2012

30. Wiwik dan Andi Sulistyo. Buku Ajar Asuhan Keperawatan Pada Klien

Dengan Gangguan Sistem Hematologi. Jakarta : Salemba Medika; 2008

31. S. Wheby M. Anemia: classification, mechanism, diagnosis, and

physiologic effects. Fundamentals of clinical hematology. 5 th ed. W. B.

Saunders Company; 1987. P . 163 – 184

32. Badham J, Zimmermann MB, Kraemer K. The Guidebook Nutritional

Anemia. SIGHT AND LIFE; 2007. P. 1 – 50

33. Wahyuni A S, Anemia Defisiensi Besi Pada Balita. Fakultas Kedokteran

USU ; 2004

34. Kharunnisa I, Syarief I, Rahmatini. Gambaran Elektrolit dan Gula Darah

Pasien Kejang Demam yang Dirawat di Bangsal Anak RSUP Dr. M.

Djamil. Jurnal Kesehatan Andalas : 2013

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35. Sumijati M.E, dkk. Asuhan Keperawatan Pada Kasus Penyakit Yang

Lazim Terjadi Pada Anak. Surabaya. PERKANI : 2000

36. Abdul Khanis. Defisiensi Besi dengan Parameter sTfR sebagai Faktor

Risiko Bangkitan Kejang Demam. Semarang. Ilmu Kesehatan Anak

Universitas Diponegoro : 2010

37. Attila D, dkk. Kejang Demam dan Faktor yang Mempengaruhi Rekurensi.

Jakarta. Sari Pediatri : 2012;14(1):57-61.

38. Daoud AS, Batieha A, Abu-Ekteish F, Gharaibeh N, Ajlouni S, Hijazi S.

Iron status: a possible risk factor or the first febrile seizure. Epilepsia

2002; 43: 740-743.

39. Kumari P, Nair MK, Nair SM, Kailas L, Geeta S. Iron deficiency as a risk

factor for simple febrile seizure--a case control study. Indian Pediatrics

2012; 49: 17-19.

40. Hartfield DS, Tan J, Yager JY, Rosychuk RJ, Spady D, Haines C, et al.

The association between iron deficiency and febrile seizures in childhood.

Clin Pediatr (Phila) 2009; 48: 420-426

Page 6: 1. Soetomenggolo TS. Kejang demam. Dalam : Soetomenggolo TS

NoUsia

(bulan)Jenis Kelamin Riwayat Keluarga Durasi kejang Frekuensi Kejang Kelompok Status Hb

1 47 P + 3 2 Kasus N

2 14 L + 5 2 Kasus N

3 6 L - 2 2 Kasus N

4 6 L - 10 3 Kasus N

5 9 L - 60 3 Kasus N

6 29 L - 5 4 Kasus N

7 48 P + 1 2 Kasus N

8 9 P - 5 2 Kasus N

9 21 L + 1 3 Kasus N

10 15 L - 3 2 Kasus N

11 10 L - 5 2 Kasus anemia

12 2 L + 3 3 Kasus N

13 5 P - 3 2 Kasus N

14 9 P - 20 1 Kasus N

15 18 L + 1 3 Kasus anemia

Page 7: 1. Soetomenggolo TS. Kejang demam. Dalam : Soetomenggolo TS

16 18 P + 1 2 Kasus anemia

17 15 L + 20 2 Kasus anemia

18 48 L - 5 2 Kasus anemia

19 17 L - 1 2 Kasus anemia

20 3 P - 1 2 Kasus anemia

21 46 P - 3 3 Kasus anemia

22 15 P + 3 2 Kasus anemia

23 19 L + 2 2 Kasus anemia

24 32 L - 20 2 Kasus anemia

25 15 P + 2 2 Kasus anemia

26 13 P - 20 2 Kasus anemia

27 7 P + 2 4 Kasus anemia

28 16 L - 5 3 Kasus anemia

29 16 L - 3 3 Kasus anemia

30 9 L + 20 2 Kasus N

31 12 P - 10 2 Kasus anemia

32 2 L + 5 2 Kasus anemia

Page 8: 1. Soetomenggolo TS. Kejang demam. Dalam : Soetomenggolo TS

33 8 L - 5 2 Kasus N

34 19 L + 2 2 Kasus N

35 29 L - 5 3 Kasus N

36 42 P + 5 2 Kasus anemia

37 55 P - 3 2 Kasus N

38 22 L - 5 2 Kasus N

39 16 L + 5 2 Kasus anemia

40 20 L - 5 3 Kasus N

41 36 L - 15 2 Kasus anemia

42 10 L + 20 1 Kasus N

43 25 L - 5 3 Kasus anemia

44 12 L + 5 2 Kasus anemia

45 25 L - 5 2 Kasus N

46 14 L + 5 2 Kasus anemia

47 6 P - 5 2 Kasus N

48 24 L - 5 1 Kontrol N

49 36 L - 2 1 Kontrol N

Page 9: 1. Soetomenggolo TS. Kejang demam. Dalam : Soetomenggolo TS

50 13 P + 5 1 Kontrol N

51 34 L + 1 1 Kontrol N

52 18 L + 5 1 Kontrol N

53 21 L - 5 1 Kontrol N

54 10 L + 1 1 Kontrol N

55 16 L - 5 1 Kontrol N

56 44 L + 5 1 Kontrol N

57 40 L + 1 1 Kontrol N

58 29 L + 5 1 Kontrol N

59 7 L - 5 1 Kontrol N

60 17 P + 10 1 Kontrol anemia

61 6 L + 5 1 Kontrol N

62 51 L - 5 1 Kontrol N

63 31 L - 5 1 Kontrol N

64 32 P + 1 1 Kontrol N

65 32 L - 2 1 Kontrol N

66 44 L + 5 1 Kontrol N

Page 10: 1. Soetomenggolo TS. Kejang demam. Dalam : Soetomenggolo TS

67 5 L - 10 1 Kontrol N

68 37 L + 2 1 Kontrol N

69 18 P - 10 1 Kontrol N

70 7 P + 5 1 Kontrol N

71 25 P - 3 1 Kontrol N

72 8 P - 5 1 Kontrol N

73 8 L - 10 1 Kontrol N

74 40 L + 3 1 Kontrol N

75 1 P + 1 2 Kontrol N

76 9 L + 1 1 Kontrol N

77 16 L - 2 1 Kontrol N

78 18 P - 10 1 Kontrol N

79 2 P - 15 1 Kontrol N

80 53 L - 2 1 Kontrol N

81 12 L + 5 1 Kontrol anemia

82 13 L + 5 1 Kontrol anemia

83 8 L - 3 1 Kontrol anemia

Page 11: 1. Soetomenggolo TS. Kejang demam. Dalam : Soetomenggolo TS

84 18 P + 3 1 Kontrol anemia

85 31 L - 5 1 Kontrol anemia

86 18 L - 5 1 Kontrol anemia

87 32 L + 5 1 Kontrol anemia

88 16 L - 5 1 Kontrol anemia

89 18 P - 10 1 Kontrol N

90 2 P - 14 1 Kontrol N

91 53 L - 2 1 Kontrol N

92 33 L - 5 1 Kontrol N

93 27 L + 5 1 Kontrol anemia

94 30 P - 5 1 Kontrol N

Page 12: 1. Soetomenggolo TS. Kejang demam. Dalam : Soetomenggolo TS

Hasil Analisis Data

1. Lampiran karakteristik dasar subyek penelitian

Jenis kelaminKelompok * Jenis Kelamin Crosstabulation

Jenis Kelamin

TotalL P

Kelompok Kasus Count 31 16 47

Expected Count 32,0 15,0 47,0

% within Kelompok 66,0% 34,0% 100,0%

% within Jenis Kelamin 48,4% 53,3% 50,0%

% of Total 33,0% 17,0% 50,0%

Kontrol Count 33 14 47

Expected Count 32,0 15,0 47,0

% within Kelompok 70,2% 29,8% 100,0%

% within Jenis Kelamin 51,6% 46,7% 50,0%

% of Total 35,1% 14,9% 50,0%

Total Count 64 30 94

Expected Count 64,0 30,0 94,0

% within Kelompok 68,1% 31,9% 100,0%

% within Jenis Kelamin 100,0% 100,0% 100,0%

% of Total 68,1% 31,9% 100,0%

Chi-Square Tests

Value df

Asymp. Sig. (2-

sided)

Exact Sig. (2-

sided)

Exact Sig. (1-

sided)

Pearson Chi-Square ,196a 1 ,658

Continuity Correctionb ,049 1 ,825

Likelihood Ratio ,196 1 ,658

Fisher's Exact Test ,825 ,413

N of Valid Cases 94

Page 13: 1. Soetomenggolo TS. Kejang demam. Dalam : Soetomenggolo TS

a. 0 cells (,0%) have expected count less than 5. The minimum expected count is 15,00.

b. Computed only for a 2x2 table Usia

Test Statisticsa

Usia

Mann-Whitney U 923,000

Wilcoxon W 2051,000

Z -1,374

Asymp. Sig. (2-tailed) ,170

a. Grouping Variable: Kelompok

Riwayat keluarga

Kelompok * Riwayat Keluarga Crosstabulation

Riwayat Keluarga

Total- +

Kelompok Kasus Count 27 20 47

Expected Count 26,5 20,5 47,0

% within Kelompok 57,4% 42,6% 100,0%

% within Riwayat Keluarga 50,9% 48,8% 50,0%

% of Total 28,7% 21,3% 50,0%

Kontrol Count 26 21 47

Expected Count 26,5 20,5 47,0

% within Kelompok 55,3% 44,7% 100,0%

% within Riwayat Keluarga 49,1% 51,2% 50,0%

% of Total 27,7% 22,3% 50,0%

Total Count 53 41 94

Expected Count 53,0 41,0 94,0

% within Kelompok 56,4% 43,6% 100,0%

% within Riwayat Keluarga 100,0% 100,0% 100,0%

% of Total 56,4% 43,6% 100,0%

Page 14: 1. Soetomenggolo TS. Kejang demam. Dalam : Soetomenggolo TS

Chi-Square Tests

Value df

Asymp. Sig. (2-

sided)

Exact Sig. (2-

sided)

Exact Sig. (1-

sided)

Pearson Chi-Square ,043a 1 ,835

Continuity Correctionb ,000 1 1,000

Likelihood Ratio ,043 1 ,835

Fisher's Exact Test 1,000 ,500

N of Valid Cases 94

a. 0 cells (,0%) have expected count less than 5. The minimum expected count is 20,50.

b. Computed only for a 2x2 table

2. Analisis anemia dengan manifestasi klinis kejang demamKelompok * Status Hb Crosstabulation

Status Hb

Totalanemia N

Kelompok 1 Count 24 23 47

Expected Count 17,0 30,0 47,0

% within Kelompok 51,1% 48,9% 100,0%

% within Status Hb 70,6% 38,3% 50,0%

% of Total 25,5% 24,5% 50,0%

2 Count 10 37 47

Expected Count 17,0 30,0 47,0

% within Kelompok 21,3% 78,7% 100,0%

% within Status Hb 29,4% 61,7% 50,0%

% of Total 10,6% 39,4% 50,0%

Total Count 34 60 94

Expected Count 34,0 60,0 94,0

% within Kelompok 36,2% 63,8% 100,0%

Page 15: 1. Soetomenggolo TS. Kejang demam. Dalam : Soetomenggolo TS

% within Status Hb 100,0% 100,0% 100,0%

% of Total 36,2% 63,8% 100,0%

Chi-Square Tests

Value df

Asymp. Sig. (2-

sided)

Exact Sig. (2-

sided)

Exact Sig. (1-

sided)

Pearson Chi-Square 9,031a 1 ,003

Continuity Correctionb 7,787 1 ,005

Likelihood Ratio 9,237 1 ,002

Fisher's Exact Test ,005 ,002

N of Valid Cases 94

a. 0 cells (0,0%) have expected count less than 5. The minimum expected count is 17,00.

b. Computed only for a 2x2 table

Durasi kejang

Status Hb * Durasi Kejang Crosstabulation

Durasi Kejang

Total< 15 menit > 15 menit

Status Hb anemia Count 31 3 34

Expected Count 31,5 2,5 34,0

% within Status Hb 91,2% 8,8% 100,0%

% within Durasi Kejang 35,6% 42,9% 36,2%

% of Total 33,0% 3,2% 36,2%

N Count 56 4 60

Expected Count 55,5 4,5 60,0

% within Status Hb 93,3% 6,7% 100,0%

% within Durasi Kejang 64,4% 57,1% 63,8%

% of Total 59,6% 4,3% 63,8%

Total Count 87 7 94

Page 16: 1. Soetomenggolo TS. Kejang demam. Dalam : Soetomenggolo TS

Expected Count 87,0 7,0 94,0

% within Status Hb 92,6% 7,4% 100,0%

% within Durasi Kejang 100,0% 100,0% 100,0%

% of Total 92,6% 7,4% 100,0%

Chi-Square Tests

Value df

Asymp. Sig. (2-

sided)

Exact Sig. (2-

sided)

Exact Sig. (1-

sided)

Pearson Chi-Square ,146a 1 ,702

Continuity Correctionb ,000 1 1,000

Likelihood Ratio ,143 1 ,705

Fisher's Exact Test ,701 ,497

N of Valid Cases 94

a. 2 cells (50,0%) have expected count less than 5. The minimum expected count is 2,53.

b. Computed only for a 2x2 table

Frekuensi kejang

Frekuensi kejang * Status Hb Crosstabulation

Status Hb

Totalanemia N

Frekuensi kejang 1,00 Count 10 38 48

Expected Count 17,4 30,6 48,0

% within Frekuensi kejang 20,8% 79,2% 100,0%

% within Status Hb 29,4% 63,3% 51,1%

% of Total 10,6% 40,4% 51,1%

2,00 Count 24 22 46

Expected Count 16,6 29,4 46,0

% within Frekuensi kejang 52,2% 47,8% 100,0%

% within Status Hb 70,6% 36,7% 48,9%

% of Total 25,5% 23,4% 48,9%

Page 17: 1. Soetomenggolo TS. Kejang demam. Dalam : Soetomenggolo TS

Total Count 34 60 94

Expected Count 34,0 60,0 94,0

% within Frekuensi kejang 36,2% 63,8% 100,0%

% within Status Hb 100,0% 100,0% 100,0%

% of Total 36,2% 63,8% 100,0%

1 = 1 x kejang dalam 24 jam

2 = 2 x kejang dalam 24 jam

Chi-Square Tests

Value df

Asymp. Sig. (2-

sided)

Exact Sig. (2-

sided)

Exact Sig. (1-

sided)

Pearson Chi-Square 9,993a 1 ,002

Continuity Correctionb 8,682 1 ,003

Likelihood Ratio 10,216 1 ,001

Fisher's Exact Test ,002 ,001

N of Valid Cases 94

a. 0 cells (0,0%) have expected count less than 5. The minimum expected count is 16,64.

b. Computed only for a 2x2 table

Page 18: 1. Soetomenggolo TS. Kejang demam. Dalam : Soetomenggolo TS

RIWAYAT HIDUP PENULIS

A. Identitas

Nama : Muhamad Helmi H

Tempat, tanggal lahir : Salatiga 15 September 1992

Agama : Islam

Jenis Kelamin : Laki-laki

Alamat : Jalan Raden Patah 145 Salatiga

B. Riwayat Pendidikan

1. TK Tarbiyatul Banin Salatiga

2. SD Negeri 01 Salatiga

3. SMP Negeri 01 Salatiga

4. SMA Negeri 01 Salatiga

C. Keterangan Keluarga

Ayah Kandung : Drs Fadholi

Ibu Kandung : Dra Anis Rosiqoh

Kakak Kandung : Happy Risdian

Adik Kandung : Muhammad Henri Indrawan

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